Sickness in mammals can lead to cognition deficits, although the underlying mechanisms remain elusive. In a recent Nature Medicine article, Garré et al. (2017) report that sickness-induced cortical dendritic spine loss and impaired memory formation is mediated by CX3CR1 monocyte-derived TNF-α.
Download full-text PDF |
Source |
|---|---|
| http://dx.doi.org/10.1016/j.immuni.2017.06.022 | DOI Listing |
Publication Analysis
Top Keywords
spineless behavior
4
behavior cx3cr1
4
cx3cr1 monocytes
4
monocytes response
4
response infection
4
infection sickness
4
sickness mammals
4
mammals lead
4
lead cognition
4
cognition deficits
4
Similar Publications
Want AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!