AI Article Synopsis
- The text indicates that there is an error in the original article associated with the DOI (Digital Object Identifier) 10.1371/journal.pone.0177046.!
- It implies that a correction has been published to address the inaccuracies found in the initial research findings or interpretations.!
- Such corrections are important for maintaining the integrity and reliability of academic literature and ensuring that readers have access to accurate information.!
Article Abstract
[This corrects the article DOI: 10.1371/journal.pone.0177046.].
Download full-text PDF |
Source |
|---|---|
| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5459558 | PMC |
| http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0179296 | PLOS |
Publication Analysis
Top Keywords
Similar Publications
The Fat-Dachsous planar polarity pathway competes with hinge contraction to orient polarized cell behaviors during Drosophila wing morphogenesis.
Curr Biol
December 2024
School of Biosciences, University of Sheffield, Firth Court, Sheffield, S10 2TN, UK. Electronic address:
During tissue morphogenesis, an interplay of biochemical pathways and mechanical cues regulates polarized cell behaviors, the balance of which leads to tissues reaching their correct shape and size. A well-studied example of a biochemical regulator is the highly conserved Fat-Dachsous (Ft-Ds) pathway that coordinates planar polarized cell behaviors and growth in epithelial tissues. For instance, in the Drosophila larval wing disc, the Ft-Ds pathway acts via the atypical myosin Dachs to control tissue shape by promoting the orientation of cell divisions primarily in a proximodistal (PD) direction.
View Article and Find Full Text PDFGeneration of Isogenic iPSC Lines for Studying the Effect of the p.N515del (c.1543_1545delAAC) Variant on MYBPC3 Function and Hypertrophic Cardiomyopathy Pathogenesis.
Int J Mol Sci
November 2024
Federal Research Centre Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, 630090 Novosibirsk, Russia.
The clinical significance of numerous cardiovascular gene variants remains to be determined. CRISPR/Cas9 allows for the introduction and/or correction of a certain variant in induced pluripotent stem cells (iPSCs). The resulting isogenic iPSC lines can be differentiated into cardiomyocytes and used as a platform to assess the pathogenicity of the variant.
View Article and Find Full Text PDFCorrection: NudCL2 regulates cell migration by stabilizing both myosin-9 and LIS1 with Hsp90.
Cell Death Dis
December 2024
Department of Cell Biology, and Institute of Gastroenterology of the Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, Zhejiang, China.
Want AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!