This article has been withdrawn at the request of the editor and publisher. The publisher regrets that an error occurred which led to the premature publication of this paper. This error bears no reflection on the article or its authors. The publisher apologizes to the authors and the readers for this unfortunate error. The article was subsequently accepted and published and can be viewed here: https://doi.org/10.1016/j.ibmb.2017.05.005 The full Elsevier Policy on Article Withdrawal can be found at http://www.elsevier.com/locate/withdrawalpolicy.
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http://dx.doi.org/10.1016/j.ibmb.2017.02.004 | DOI Listing |
PLoS One
April 2019
Department of Biochemistry, Jawaharlal Institute of Post-graduate Medical Education and Research (JIPMER), Puducherry, India.
Platelet activation has been reported to play a major role in inflammatory response and thrombocytopenia during dengue viral infection. Cells expressing FcϒR2Aand DC-SIGN receptors are reported to be involved in dengue virulence. The present study is designed to assess the expression level of these two receptors on platelet surface collected from dengue patients and to study its association in patients with platelet RNA positive for dengue virus.
View Article and Find Full Text PDFAsian-Australas J Anim Sci
May 2018
Faculty of Agriculture, Department of Animal Science, Lorestan University, Beijing, China.
Objective: This study was conducted to investigate hematological and vasodilator characteristics for high altitude acclimatization in Holstein heifers ascended to high altitude.
Methods: Holstein heifers resided at low altitude (LH; Beijing, China 43 meter), and Holstein heifers ascended to high altitude (Lhasa city, Tibet; 3,658 meter) after one year exposure including hypoxia acclimated Holstein heifers (AH; with mean pulmonary arterial pressure mPAP < 41 mmHg) and Holstein heifers affected by high altitude pulmonary hypertension (HAPH; with mPAP > 49 mmHg) and crossbred cattle (CB; Holstein × Tibet cattle) born and resided in Tibet were selected to compare parameters of blood gas, blood cells and vasodilators. Each group included 15 animals with 16 to 18 month of age.
Psychopharmacology (Berl)
July 2018
Department of Pharmacology and Toxicology, Medical College of Virginia Campus, Virginia Commonwealth University, Richmond, VA, USA.
Rationale: Recent preclinical data has implicated the α7 nicotinic acetylcholine receptor (nAChR) as a target in modulating nicotine reward. However, the role of the channel properties of the α7 nAChR in nicotine withdrawal is unknown.
Objectives: This study aimed to investigate the impact of α7 nAChR pharmacological modulation on mecamylamine-precipitated nicotine withdrawal behaviors in mice by using positive allosteric modulators (PAMs).
Insect Biochem Mol Biol
February 2017
Hubei Key Laboratory of Genetic Regulation and Integrative Biology, School of Life Sciences, Central China Normal University, Wuhan 430079, China.
This article has been withdrawn at the request of the editor and publisher. The publisher regrets that an error occurred which led to the premature publication of this paper. This error bears no reflection on the article or its authors.
View Article and Find Full Text PDFImmunol Res
January 2014
Neuroimmunology Research Unit, The Sagol School of Neuroscience, The School of Psychological Sciences, Tel Aviv University, Room #113b, Sharett Building, Tel Aviv, 69978, Israel,
To test whether marginating-pulmonary (MP) leukocytes in mice have a unique potential to identify and destroy aberrant circulating cells, we compared MP to circulating leukocytes with respect to natural killer (NK) cytotoxicity, proinflammatory characteristics, molecular determinants of activation, and response to IL-12 immunostimulation. Cytotoxicity was assessed employing the YAC-1, B16F10, and 3LL target lines. C57BL/6 mice were injected with either saline or murine IL-12 (0.
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