AI Article Synopsis

  • Tocotrienols (T3), particularly δ-T3 and γ-T3, show promise in enhancing insulin gene expression and secretion in rat pancreatic cells, especially when glucose levels are elevated.
  • Short-term exposure to δ- and γ-T3 significantly increases preproinsulin mRNA levels, indicating their role in insulin synthesis, unlike α-T3 which is less effective.
  • The addition of potassium chloride (KCl) further boosts insulin production, highlighting the potential of δ-T3 in regulating insulin synthesis in a glucose-rich environment.

Article Abstract

Tocotrienols (T3) are well-known for their antioxidant properties besides showing therapeutic potential in clinical complications such as hyperlipidemia induced by diabetes. The aim of this study was to determine the effects of δ-T3, γ-T3, and α-T3 on insulin secretion-associated genes expression of rat pancreatic islets in a dynamic culture. Pancreatic islets freshly isolated from male Wistar rats were treated with T3 for 1 h at 37°C in a microfluidic system with continuous operation. The cells were collected for total RNA extraction and reverse-transcribed, followed by measurement of insulin secretion-associated genes expression using quantitative real-time polymerase chain reaction. Molecular docking experiments were performed to gain insights on how the T3 bind to the receptors. Short-term exposure of δ- and γ-T3 to pancreatic β cells in a stimulant glucose condition (16.7 mM) significantly regulated preproinsulin mRNA levels and insulin gene transcription. In contrast, α-T3 possessed less ability in the activation of insulin synthesis level. Essentially, potassium chloride (KCl), a β cell membrane depolarising agent added into the treatment further enhanced the insulin production. δ- and γ-T3 revealed significantly higher quantitative expression in most of the insulin secretion-associated genes groups containing 16.7 mM glucose alone and 16.7 mM glucose with 30 mM KCl ranging from 600 to 1200 μM (p < 0.05). The findings suggest the potential of δ-T3 in regulating insulin synthesis and glucose-stimulated insulin secretion through triggering pathway especially in the presence of KCl.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5003849PMC
http://dx.doi.org/10.3389/fphar.2016.00291DOI Listing

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