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FIRS: Neonatal considerations.
Semin Fetal Neonatal Med
August 2020
Loyola University Medical Center, 2160 South 1st Avenue, Division of Neonatal and Perinatal Medicine, Maywood, IL, 60153, USA.
Fetal Inflammatory Response Syndrome (FIRS) is the fetal counterpart of systemic inflammatory response syndrome (SIRS) described in adults. When the fetus is directly exposed to inflammation of the fetal membranes or the placental-fetal circulation, and organs are adversely affected, the disorder is known as FIRS. This syndrome can significantly affect multiple organs with significant short and long term implications for the newborn.
View Article and Find Full Text PDFABO blood group and procoagulant factors: the hypercoagulation hypothesis ABO and Procoagulant Factors.
Pediatr Res
September 2019
Department of Pediatrics - Neonatology, Loyola University Medical Center, Maywood, IL, USA.
Background: The correlation between procoagulant levels-factor VIII (FVIII), von Willebrand factor (vWF), and fibrinogen-and risk of thrombosis has been well documented in adult populations. We hypothesize that interaction of passively transferred isoagglutinins in premature neonates with a compromised immune system may trigger an immune response that can target the immature gastrointestinal tract. The objective of this study is to evaluate if there are procoagulant level differences in preterm newborns stratified by ABO blood group.
View Article and Find Full Text PDF[C. Racinet et al. in response to the correspondence by J. Muraskas and by E. Saling about the article by C. Racinet et al.: Neonatal metabolic acidosis at birth: In search of a reliable marker. Gynecol Obstet Fertil 2016;44:357-62].
Gynecol Obstet Fertil Senol
April 2017
University of Ottawa, Ottawa, Canada; Department of Pediatrics, Children's Hospital of East Ontario, Ottawa, Canada.
JK Muraskas in response to the article by C. Racinet et al. Neonatal metabolic acidosis at birth: In search of a reliable marker. Gynecol Obstet Fertil 2016; 44: 357-62.
Gynecol Obstet Fertil
September 2017
Loyola University Medical Center, Loyola University Stritch School of Medicine, Neonatal and Maternal Fetal Medicine, 2160 S, 1st avenue, Russo Surgical Pavilion, Maywood, Illinois 60153, United States. Electronic address:
The role of fetal inflammatory response syndrome and fetal anemia in nonpreventable term neonatal encephalopathy.
J Perinatol
May 2016
Department of Obstetrics and Gynecology and Pediatrics, University of Mississippi Medical Center, Jackson, MS, USA.
Objective: To evaluate asphyxial patterns in term encephalopathic newborns caused by chorioamnionitis or intrapartum blood loss that resulted in cerebral palsy and allegations of obstetrical professional liability.
Study Design: As an expert witness, JKM identified term newborns with profound neurologic impairment: 18 born in the presence of chorioamnionitis and 14 with significant anemia.
Result: In both study groups, profound depression with low 10-min Apgars was associated with early-onset seizures (88%), multiorgan failure (94%) and a partial prolonged injury to the cortex and subcortical white matter (94%).
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