Activity of lecithine-cholesterol acyltransferase (LCAT) was studied in blood of 26 patients with atherosclerosis of various localization after course of treatment involving three-fold hemosorption. The initial activity of the enzyme, decreased distinctly as compared with normal state, was dissimilarly altered during hemosorption. The system LCAT-HDLP was stabilized after the third hemosorption. These results relevant to programming hemosorption therapy of patients with atherosclerosis.
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