Download full-text PDF |
Source |
|---|---|
| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5331405 | PMC |
| http://dx.doi.org/10.14744/AnatolJCardiol.2016.7130 | DOI Listing |
Publication Analysis
Top Keywords
Similar Publications
Vitamin D Dependent Rickets 2A With Alopecia: Three Cases With Novel Genetic Variants.
Pediatr Dermatol
December 2024
Paediatric Endocrinology Division, Department of Paediatrics, Christian Medical College Vellore, Vellore, Tamil Nadu, India.
Vitamin D-dependent rickets type 2A (VDDR2A) is a rare cause of infantile-onset alopecia, characterized by severe hypotrichosis, small cutaneous cysts, early-onset treatment-resistant rickets, and hypocalcemia. Alopecia, often starting a few weeks to months after birth, may be the presenting feature. We present three cases of VDDR2A with genetic variants in the vitamin D receptor (VDR) gene, their clinical features and biochemical parameters.
View Article and Find Full Text PDFPlasmapheresis for extracorporeal membrane oxygenation (ECMO)-induced hemolysis in infants.
J Extra Corpor Technol
December 2024
Division of Pediatric Nephrology, Joe DiMaggio Children's Hospital, 1131 N35th Ave, Hollywood, FL 33021, USA - Charles E. Schmidt College of Medicine at Florida Atlantic University, 777 Glades Rd BC-71, Boca Raton, FL 33431, USA.
Background: Intravascular hemolysis is a known complication of extracorporeal membrane oxygenation (ECMO). Characterized by elevated plasma-free hemoglobin (PFH), intravascular hemolysis is associated with cytotoxic effects leading to renal replacement therapy (RRT), longer ECMO runs, and mortality. Therapeutic plasma exchange (TPE) in tandem with ECMO was described as a therapy for various pathologic conditions, but there are no Extracorporeal Life Support Organization (ELSO) guidelines for the treatment of ECMO-induced hemolysis.
View Article and Find Full Text PDFAn electrolyte disturbance masquerading as a panic attack.
JAAPA
January 2025
Anne Wildermuth is an assistant professor in the School of Medicine at the Uniformed Services University in Bethesda, Md., and practices in emergency medicine with MedStar Emergency Physicians in Clinton, Md. At the time this article was written, Meagan Lantz and Erin Sagers were students in the PA program at the George Washington University in Washington, D.C. Ms. Lantz now practices in cardiothoracic surgery at Stanford Health Care in Palo Alto, Calif. Ms. Sagers now practices in orthopedics at EmergeOrtho Coastal Region in Wilmington, N.C. The authors have disclosed no potential conflicts of interest, financial or otherwise. The views expressed in this article are those of the authors and not of the Department of Defense, Henry M. Jackson Foundation for the Advancement of Military Medicine, Inc., or other federal agencies.
Because proton pump inhibitors (PPIs) are so commonly used, their safety and potential adverse reactions often are not considered. This article describes a patient whose paresthesias and severe muscle cramps were attributed to panic attacks but actually were caused by hypocalcemia secondary to PPI-induced hypomagnesemia. This case serves as a reminder that PPIs should be used cautiously and appropriately with consideration for regular monitoring of electrolytes and vitamin B12.
View Article and Find Full Text PDFSevere hypophosphataemia and hypocalcaemia following intravenous ferric derisomaltose and denosumab administration.
BMJ Case Rep
December 2024
Division of Endocrinology, Department of Medicine, Jewish General Hospital, McGill University, Montreal, Quebec, Canada.
Serum calcium and phosphorus levels are tightly regulated by the calciotropic hormone parathyroid hormone, fibroblast growth factor 23 and 1,25(OH) vitamin D. Commonly prescribed therapies for iron-deficiency anaemia (IDA) such as ferric carboxymaltose and ferric derisomaltose (FDM) have been shown to disrupt phosphorus homeostasis, resulting in hypophosphataemia. Similarly, denosumab use can result in hypocalcaemia due to the inhibition of osteoclastic maturation, activity and survival.
View Article and Find Full Text PDFST-Segment Elevation: Hypocalcemia or Takotsubo Syndrome? A Diagnostic Dilemma.
JACC Case Rep
December 2024
Cardiology Unit, Interdisciplinary Department of Medicine, University of Bari Aldo Moro, University Hospital Consortium, Polyclinic of Bari, Bari, Italy.
A wide range of etiologies, both ischemic and nonischemic, can produce an electrocardiographic pattern of ST-segment elevation (STE), including Takotsubo syndrome (TTS) and electrolyte imbalances. Instances of hypocalcemia-induced TTS and STE are exceedingly rare in medical literature. This paper presents the case of a 75-year-old woman with advanced ovarian cancer and no prior heart issues, who exhibited diffuse STE on electrocardiogram, resembling acute coronary syndrome.
View Article and Find Full Text PDFWant AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!