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Totally intrabiliary colorectal liver metastasis mimicking intraductal growth-type cholangiocarcinoma.
Updates Surg
June 2016
Division of General and Hepatobiliary Surgery, Department of Surgery G.B. Rossi University Hospital, University of Verona, Piazzale L.A. Scuro, 10, 37134, Verona, Italy.
Intraductal papillary cholangiocarcinoma and atypical biliary epithelial lesions confused with intrabiliary extension of metastatic colorectal carcinoma.
Hepatogastroenterology
August 2007
Department of Human Pathology, Kanazawa University Graduate School of Medicine, Kanazawa 920-8640, Japan.
Intrabiliary metastasis of colorectal carcinoma is not infrequent. We demonstrated interesting intraductal biliary epithelial lesions in two cases of colorectal carcinoma. The first case (65-year-old female) presented intrahepatic papillary adenocarcinoma with intrabiliary extension.
View Article and Find Full Text PDFS-adenosylmethionine protects against intrabiliary glutathione degradation induced by long-term administration of cyclosporin A in the rat.
Toxicology
September 2004
Department of Physiology and Pharmacology, University of Salamanca, 37071 Salamanca, Spain.
We investigate the ability of S-adenosylmethionine (SAMe) to antagonize the cyclosporine A (CyA)-induced inhibition of biliary glutathione efflux induced by long-term administration of CyA (10 mg/kg per day-CyA10 or 20 mg/kg per day-CyA20 for 4 weeks) in rats. CyA treatment reduced the liver content of total glutathione and caused a significant increase in the oxidized-to-reduced glutathione ratio and the thiobarbituric acid-reactive substances (TBARS) concentration. When the rats were concurrently treated with SAMe (10 mg/kg twice daily) and CyA, all these parameters did not significantly differ from control values.
View Article and Find Full Text PDFEffects of S-adenosylmethionine on intrabiliary glutathione degradation induced by long-term administration of cyclosporin A in the rat.
Toxicology
July 2004
Department of Physiology and Pharmacology, University of Salamanca, 37071 Salamanca, Spain.
We investigate the ability of S-adenosylmethionine (SAMe) to antagonize the cyclosporine A (CyA)-induced inhibition of biliary glutathione efflux induced by long-term administration of CyA (10 mg/kg per day-CyA(10) or 20 mg/kg per day-CyA(20) for 4 weeks) in rats. CyA treatment reduced the liver content of total glutathione and caused a significant increase in the oxidized-to-reduced glutathione ratio and the thiobarbituric acid-reactive substances (TBARS) concentration. When the rats were concurrently treated with SAMe (10 mg/kg twice daily) and CyA, all these parameters did not significantly differ from control values.
View Article and Find Full Text PDFSurgical management of spontaneous intrabiliary rupture of hydatid liver cysts.
Surg Today
September 2002
Department of General Surgery, University of Erciyes, School of Medicine, 38039 Kayseri, Turkey.
Purpose: The most common complication of hydatid liver cysts is spontaneous rupture into the biliary tract. This study was conducted to evaluate the surgical management of spontaneous intrabiliary rupture of a hydatid liver cyst in 41 patients.
Methods: The preoperative diagnosis was confirmed by ultrasound in all 41 patients, 37 of whom were jaundiced.
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