A great boost to research into LP aetiopathogenesis has been forthcoming from recent studies that define its connection with chronic hepatopathies. This subject is addressed by giving considerable room to these new research approaches, although other aspects, such as the relationship with dental materials, are not neglected. The proposal of a model of aetiopathogenetic mechanism obtained by analysis of the data supplied by bibliographic research furnishes a contribution to the subject consisting essentially of giving an overall, omni-inclusive view of recent findings on the aetiopathogenesis of LP and in connecting these significantly for the purpose of obtaining better understanding of the physiopathology of the disease.
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