Stress is a major problem of our society, as it is the cause of many health problems and huge economic losses in companies. Continuous high mental workloads and non-stop technological development, which leads to constant change and need for adaptation, makes the problem increasingly serious for office workers. To prevent stress from becoming chronic and provoking irreversible damages, it is necessary to detect it in its early stages. Unfortunately, an automatic, continuous and unobtrusive early stress detection method does not exist yet. The multimodal nature of stress and the research conducted in this area suggest that the developed method will depend on several modalities. Thus, this work reviews and brings together the recent works carried out in the automatic stress detection looking over the measurements executed along the three main modalities, namely, psychological, physiological and behavioural modalities, along with contextual measurements, in order to give hints about the most appropriate techniques to be used and thereby, to facilitate the development of such a holistic system.
Download full-text PDF |
Source |
---|---|
http://dx.doi.org/10.1016/j.jbi.2015.11.007 | DOI Listing |
Med Educ Online
December 2025
Imperial College School of Medicine, Imperial College London, London, UK.
Resitting, being offered a 'second chance' at an exam following failure to achieve a passing grade, is both common and stressful in medical school. There is a significant gap in the medical education literature around evidence-based support for resitting medical students. The study explores medical student experiences of resits through a peer-assisted learning programme (PAL) delivered to early years resitting medical students at Imperial College School of Medicine (ICSM) in 2021 and 2022.
View Article and Find Full Text PDFBMJ Open
March 2025
Clinical College of Nanjing Medical University, Nanjing Drum Tower Hospital, Nanjing, Jiangsu, China
Objectives: Patients experience significant physical and psychological changes within the first 3 months post-surgery, yet few studies focus on patient experiences during the early postoperative period. This study aimed to explore the patient experiences and expectations for nursing follow-up during the home recovery period following metabolic and bariatric surgery.
Design: A qualitative descriptive study design was used.
Plant Sci
March 2025
State Key Laboratory for Conservation and Utilization of Subtropical Agro-Bioresources, Guangxi Key Laboratory for Agro-Environment and Agro-Product Safety, National Demonstration Center for Experimental Plant Science Education, College of Agriculture, Guangxi University, Nanning, 530004; Guangxi, China. Electronic address:
The GIBBERELLIN INSENSITIVE DWARF1 (GID1) gene encodes a receptor integral to Gibberellic acid (GA) signaling, which is pivotal for plant growth, development, and stress responses. Until now, GID1 genes have not been documented in mango. In this research, the mango (Mangifera indica) genome yielded four GID1 homologous genes, and this study focuses on the research of MiGID1A and MiGID1B genes.
View Article and Find Full Text PDFAgeing Res Rev
March 2025
Department of Internal Medicine, Texas Tech University Health Sciences Center, Lubbock, TX 79430. Electronic address:
A key molecule in cellular metabolism, citrate is essential for lipid biosynthesis, energy production, and epigenetic control. The etiology of Alzheimer's disease (AD), a progressive neurodegenerative illness marked by memory loss and cognitive decline, may be linked to dysregulated citrate transport, according to recent research. Citrate transporters, which help citrate flow both inside and outside of cells, are becoming more and more recognized as possible participants in the molecular processes underlying AD.
View Article and Find Full Text PDFInt Immunopharmacol
March 2025
School of Integrative Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin, China. Electronic address:
Atherosclerosis is a chronic inflammatory disease in which mitochondrial DNA (mtDNA) has emerged as a key contributor to its pathogenesis. We synthesized evidence from experimental and clinical studies showing that mtDNA damage, release, and mutation profoundly affect endothelial cells, macrophages, and vascular smooth muscle cells, thereby driving plaque initiation and progression. By activating immune signaling pathways-including cGAS-STING, NLRP3 inflammasome, and TLR9-mtDNA amplifies inflammation and oxidative stress, exacerbating atherosclerotic lesion development.
View Article and Find Full Text PDFEnter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!