A 7-month-old Irish Setter underwent transcatheter therapy of a muscular ventricular septal defect (VSD) and pulmonary valve stenosis. Standard devices for muscular VSD closure could not span the interventricular septum due to right ventricular hypertrophy, and an Amplatzer post-infarction muscular VSD occluder with a wider waist was successfully implanted. Following VSD closure, inflation of the balloon dilation catheter during balloon pulmonary valvuloplasty resulted in iatrogenic embolization of the VSD occluder to the left ventricular outflow tract. Retrieval and reimplantation of the device was achieved using a snare catheter. This report describes a potential complication and management during intracardiac device implantation in a dog. Additionally, the case illustrates that the Amplatzer post-infarction muscular VSD occluder holds potential value in animals with a hypertrophied interventricular septum that cannot be spanned using a conventional device.

Download full-text PDF

Source
http://dx.doi.org/10.1016/j.jvc.2015.08.003DOI Listing

Publication Analysis

Top Keywords

muscular vsd
12
vsd occluder
12
iatrogenic embolization
8
ventricular septal
8
septal defect
8
vsd closure
8
interventricular septum
8
amplatzer post-infarction
8
post-infarction muscular
8
vsd
6

Similar Publications

We report the case of a 3-year-old asymptomatic girl (12 kg, 96 cm) who was diagnosed with a large iatrogenic left ventricular pseudoaneurysm (LVP) on follow-up ultrasound, 14 months after apical muscular ventricular septal defect (VSD) closure with a 10 mm Amplatzer Muscular VSD occluder (Abbott, USA) due to device erosion. The LVP was successfully occluded using detachable Penumbra coils, with complete thrombo-exclusion confirmed at 12-month follow-up.

View Article and Find Full Text PDF

Aims: In repaired tetralogy of Fallot (rTOF), the septal anatomical isthmuses (AI), AI 3, between the ventricular septal defect (VSD) and pulmonary annulus, and AI 4, between the VSD and tricuspid annulus, are important ventricular tachycardia (VT) substrates when slow conducting. Our aim was to assess the influence of VSD characteristics, specifically the presence of muscular or fibrous tissue at its border, on the presence or absence of septal AIs, potentially related to VT.

Methods And Results: All consecutive rTOF patients who underwent electroanatomical mapping between January 2005 and March 2023 with an available surgical report providing VSD details (n = 91) were included.

View Article and Find Full Text PDF

Diagnosis and Management of Ventricular Septal Defects.

Rev Cardiovasc Med

November 2024

Children's Heart Institute, UT Health McGovern Medical School, Houston, TX 77030, USA.

This review addresses the diagnosis and management of ventricular septal defects (VSDs). The VSDs are classified on the basis of their size, their number, and their location in the ventricular septum. Natural history of VSDs includes spontaneous closure, development of pulmonary hypertension, onset of infundibular obstruction, and progression to aortic insufficiency.

View Article and Find Full Text PDF
Article Synopsis
  • Multiple muscular ventricular septal defects (VSDs) are challenging to identify and repair surgically, leading to potential residual defects, but 3D-printed models can improve surgical planning by accurately visualizing heart anatomy.
  • The study analyzed the use of 3D models in six pediatric patients with multiple VSDs from September 2021 to July 2023, highlighting successful surgeries performed with no postoperative complications.
  • Results showed that all patients had normal heart function during follow-up, indicating that 3D printing is a safe and effective tool for closing multiple muscular VSDs in children.
View Article and Find Full Text PDF

Voltage-gated sodium (Na) channels are pivotal for cellular signaling and mutations in Na channels can lead to excitability disorders in cardiac, muscular, and neural tissues. A major cluster of pathological mutations localizes in the voltage-sensing domains (VSDs), resulting in either gain-of-function (GoF), loss-of-function (LoF) effects, or both. However, the mechanism behind this functional divergence of mutations at equivalent positions remains elusive.

View Article and Find Full Text PDF

Want AI Summaries of new PubMed Abstracts delivered to your In-box?

Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!