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Postimplantation syndrome (PIS) is a known inflammatory response following endovascular stent placement, yet comparative data between venous and arterial stenting remains limited. This study seeks to evaluate the incidence, characteristics, and clinical implications of PIS across these two distinct vascular territories. We retrospectively analyzed 191 patients who underwent either venous (n = 36) or arterial (n = 155) stent placement.

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Background: The most common complication after endovascular aortic aneurysm repair (EVAR) is continuous sac perfusion, known as endoleak. Evaluating markers released from the aneurysm wall into circulation has been suggested as a possible alternative for detecting endoleaks. The aim of this study was to examine whether circulating concentrations of different proinflammatory interleukins (IL-1β, IL-2, IL-6, IL-8), monocyte chemoattractant protein-1 (MCP-1), and tumor necrosis factor alpha (TNF-α) were elevated in patients with endoleak after EVAR.

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  • - Type II endoleaks are common after aortic stent graft procedures, often requiring treatment such as transarterial or trans-caval embolization, but complications like infections can arise, as highlighted in a unique case study.
  • - A 71-year-old male developed an infected Gore Excluder stent graft following trans-caval embolization for a type II endoleak, which was complicated by rapid sac enlargement and difficult-to-diagnose inflammation.
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  • - Post Implantation Syndrome (PIS) affects some patients after endovascular aortic repair (EVAR), with a study of 76 patients revealing that 22% developed PIS, while the rest did not, indicating a potential link to systemic inflammatory response syndrome (SIRS).
  • - The study compared various factors, including platelet count and thrombus volume, between PIS and non-PIS groups; results showed higher baseline platelet counts and larger drops in platelet counts among PIS patients but no significant differences in aneurysm volumes.
  • - The findings suggest that platelets are key indicators for developing PIS post-EVAR, highlighting the need for further research to clarify the relationship between platelets and thrombus volumes in this context
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Abdominal aortic aneurysm (AAA) is a progressive dilatation of the aorta that can lead to aortic rupture. The pathophysiology of the disease is not well characterized but is known to be caused by the general breakdown of the extracellular matrix within the aortic wall. In this comprehensive literature review, all current research on proteins that have been investigated for their potential prognostic capabilities in patients with AAA was included.

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