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Predicting intradialytic hypotension in critically ill patients undergoing intermittent hemodialysis: a prospective observational study.
Intensive Care Med Exp
September 2024
Nephrology Division, University of California, San Diego, USA.
Background: Hypotension during dialysis arises from vasomotor tone alterations and hypovolemia, with disrupted counterregulatory mechanisms in acute kidney injury (AKI) patients. This study investigated the predictive value of preload dependency, assessed by the passive leg raising (PLR) test, and arterial tone, measured by dynamic elastance (Eadyn), for intradialytic hypotension (IDH).
Methods: In this prospective observational study conducted in a tertiary hospital ICU, hemodynamic parameters were collected from critically ill AKI patients undergoing intermittent hemodialysis using the FloTrac/Vigileo system.
The Validity of Eadyn in Spontaneously Breathing Patients.
Anesth Analg
November 2015
Department of Anesthesiology, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands, Department of Anesthesiology and Intensive Care Medicine, Maria Middelares Hospital, Ghent, Belgium.
Dynamic arterial elastance as a predictor of arterial pressure response to fluid administration: a validation study.
Crit Care
November 2014
Department of Intensive Care Medicine, St George's Healthcare NHS Trust and St George's University of London, Blackshaw Road, Tooting, London, SW17 0QT, UK.
Introduction: Functional assessment of arterial load by dynamic arterial elastance (Eadyn), defined as the ratio between pulse pressure variation (PPV) and stroke volume variation (SVV), has recently been shown to predict the arterial pressure response to volume expansion (VE) in hypotensive, preload-dependent patients. However, because both SVV and PPV were obtained from pulse pressure analysis, a mathematical coupling factor could not be excluded. We therefore designed this study to confirm whether Eadyn, obtained from two independent signals, allows the prediction of arterial pressure response to VE in fluid-responsive patients.
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