In this issue of Blood, Gunasekera et al provide evidence that the high rate of factor VIII (FVIII) inhibitors seen in black hemophilia A (HA) patients is not due to a mismatch between the structure of treatment products and FVIII genotypes common in blacks.
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Source |
|---|---|
| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4556233 | PMC |
| http://dx.doi.org/10.1182/blood-2015-02-625574 | DOI Listing |
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