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The gatekeeper images in hypertrophic cardiomyopathy: the role of native T1 mapping in Anderson-Fabry disease.
Monaldi Arch Chest Dis
December 2024
Cardiology Division, Regina Montis Regalis Hospital, ASLCN1, Mondovì.
We presented a case of a 49-year-old presenting with atypical chest pain and hypertrophic phenotype cardiomyopathy without coronary artery disease. At cardiac magnetic resonance (CMR), the left ventricle was of normal volumes and preserved global ejection fraction with an asymmetric wall hypertrophy. The evaluation of native myocardial T1 has been calculated at an average global value of 924 ms, compatible with hypertrophic phenotype cardiomyopathy with reduced native T1 values as observed in Anderson-Fabry disease.
View Article and Find Full Text PDFAssessing Acute Pericarditis with T1 Mapping: A Supportive Contrast-Free CMR Marker.
Tomography
November 2024
Department of Radiology, Azienda Ospedaliero Universitaria (A.O.U.), di Cagliari-Polo di Monserrato s.s. 554 Monserrato, 09045 Cagliari, Italy.
Objective: The purpose of this study was to explore the impact of pericardial T1 mapping as a potential supportive non-contrast cardiovascular magnetic resonance (CMR) parameter in the diagnosis of acute pericarditis. Additionally, we investigated the relationship between T1 mapping values in acute pericarditis patients and their demographic data, cardiovascular risk factors, clinical parameters, cardiac biomarkers, and cardiac function.
Method: This retrospective study included CMR scans in 35 consecutive patients with acute pericarditis (26 males, 45.
Diagnostic accuracy of texture analysis applied to T- and T-Relaxation maps for liver fibrosis classification via machine-learning algorithms with liver histology as reference standard.
Eur J Radiol
December 2024
Division of Endocrinology and Metabolism, Department of Medicine III, Medical University of Vienna, Austria.
Objectives: To explore texture analysis' ability on T and T relaxation maps to classify liver fibrosis into no-to-mild liver fibrosis (nmF) versus severe fibrosis (sF) group using machine learning algorithms and histology as reference standard.
Materials And Methods: In this single-center study, patients undergoing 3 T MRI who also had histology examination were retrospectively enrolled. SNAPSHOT-FLASH sequence for T1 mapping, radial turbo-spin-echo sequence for T2 mapping and spin-echo echo-planar-imaging magnetic resonance elastography (MRE) sequences were analyzed.
Left ventricle remodeling by CMR in treated patients with primary aldosteronism and primary systemic arterial hypertension.
PLoS One
December 2024
Instituto do Coração (InCor), Hospital das Clinicas HCFMUSP, Faculdade de Medicina, Universidade de Sao Paulo, Sao Paulo, São Paulo, Brazil.
Background: Increased cardiac after load and multiple non-hemodynamic stimuli implicate in adverse left ventricular remodeling (LVR). This is particularly identifiable in treatment-resistant and secondary hypertension contexts, like primary hyperaldosteronism (PA), however little data exists on post-treatment residual LVR in these individuals.
Methods: Cardiac magnetic resonance (CMR) with T1 mapping were performed in 14 patients with treated PA matched with 15 treated patients with primary hypertension (PH) and 15 healthy individuals.
Coexistent pleural effusion is found to be associated with aggravated subclinical myocardial injury in systemic lupus erythematous using cardiovascular magnetic resonance imaging.
Front Immunol
December 2024
Department of Rheumatology and Immunology, Chengdu Fifth People's Hospital, Chengdu, China.
Article Synopsis
- The study investigates the impact of pleural effusion (PE) on cardiac function in patients with systemic lupus erythematosus (SLE), as PE is common in SLE and linked to disease activity.
- It included 111 SLE patients, revealing that 23.42% had PE, which correlated with higher inflammatory markers and more cardiac involvement as shown by multi-parameter cardiovascular magnetic resonance imaging (CMR).
- Ultimately, SLE patients with PE exhibited significantly altered cardiac metrics, such as increased late gadolinium enhancement (LGE), Native T1, extracellular volume (ECV), and T2 values compared to those without PE and healthy controls.
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