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| http://dx.doi.org/10.1136/bcr-2014-208594 | DOI Listing |
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The interplay of sleep deprivation, ferroptosis, and BACH1 in cardiovascular disease pathogenesis.
Tissue Cell
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Key Laboratory of Xin'an Medicine, Ministry of Education, Anhui Province Key Laboratory of R&D of Chinese Medicine, Anhui University of Chinese Medicine, Hefei, China. Electronic address:
Background And Objective: Sleep deprivation (SD) can induce cardiac dysfunction, myocardial injury, and sudden death. The purpose of this study is to investigate whether the mechanism of sleep deprivation induced myocardial injury is related to abnormal expression of BACH1 and ferroptosis of myocardial cells.
Methods: Thirty-six male C57BL/6 J mice were used in the study.
Performance against standardization recommendations for outpatient care of common forms of congenital heart disease.
Cardiol Young
March 2025
Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, USA.
Background: The American College of Cardiology has published clinical practice algorithms for common congenital heart lesions, including atrial septal defect, patent ductus arteriosus, valvar pulmonary stenosis, aortic coarctation, and ventricular septal defect. The purpose of this study was to define the current practice patterns in the management of these lesions and describe the impact of departure from these recommendations.
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Neonatal Congenital Heart Laboratory, Department of Pediatrics, David Geffen School of Medicine, University of California, Los Angeles, CA 90095, USA.
One out of every hundred live births present with congenital heart abnormalities caused by the aberrant development of the embryonic cardiovascular system. The conserved zinc finger transcription factor proteins, which include GATA binding protein 5 (GATA5) and GATA binding protein (GATA6) play important roles in embryonic development and their inactivation may result in congenital heart defects (CHDs). In this study, we performed genotypic-phenotypic analyses in two families affected by right-sided CHD diagnosed by echocardiography imaging.
View Article and Find Full Text PDFCrip2 affects vascular development by fine-tuning endothelial cell aggregation and proliferation.
Cell Mol Life Sci
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College of Marine Life Sciences, Key Laboratory of Evolution & Marine Biodiversity (Ministry of Education), Institute of Evolution & Marine Biodiversity, Ocean University of China, Qingdao, 266003, China.
Endothelial cell adhesion and migration are crucial to various biological processes, including vascular development. The identification of factors that modulate vascular development through these cell functions has emerged as a prominent focus in cardiovascular research. Crip2 is known to play a crucial role in cardiac development, yet its involvement in vascular development and the underlying mechanism remains elusive.
View Article and Find Full Text PDF[Diagnostic characterization and morbi-mortality in pediatric patients with diagnosis of vascular ring].
Arch Cardiol Mex
March 2025
Departamento de Cardiología Pediátrica, Instituto Nacional de Cardiología Ignacio Chávez, Ciudad de México, México.
Objective: To analyze the clinical characteristics and natural history of pediatric patients with a diagnosis of vascular ring.
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Results: Of the total, 60 (55%) were female and 50 (45%) were male.
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