AI Article Synopsis

  • The study observed that in patients with acute myocardial infarction, the kallikrein-kinin system was activated, indicating a potential physiological response to the condition.
  • Changes in the coagulation system suggested a state of hypercoagulation, which may complicate recovery.
  • The alterations in these systems during rehabilitation therapy imply a connection between their activation mechanisms and the kinin system's role in regulating blood clotting.

Article Abstract

In 161 patients with acute myocardial infarction, the kallikrein-kinin system was found to be activated. In addition, there were alterations in indices of the coagulative system, which were suggestive of hypercoagulation. The unidirectional changes in the indices of the systems in question during therapy in the periods of rehabilitation are attributable to the common character of trigger mechanisms responsible for their activation and to the involvement of the kinin system in the regulation of hemostatic potential.

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