AI Article Synopsis

  • Regulatory T (Treg) cells are crucial for maintaining immune tolerance to self and preventing autoimmune responses.
  • The study indicates that Treg cells can induce anergy (a state of unresponsiveness) in self-reactive human CD8(+) T cells, likely by influencing antigen-presenting cells' costimulatory activity.
  • The presence of anergic T cells, which have a naïve phenotype and higher levels of inhibitory markers, was detected in healthy individuals, demonstrating a potential mechanism for maintaining self-tolerance against autoimmune conditions like vitiligo.

Article Abstract

Immunological tolerance to self requires naturally occurring regulatory T (Treg) cells. Yet how they stably control autoimmune T cells remains obscure. Here, we show that Treg cells can render self-reactive human CD8(+) T cells anergic (i.e., hypoproliferative and cytokine hypoproducing upon antigen restimulation) in vitro, likely by controlling the costimulatory function of antigen-presenting cells. Anergic T cells were naïve in phenotype, lower than activated T cells in T cell receptor affinity for cognate antigen, and expressed several coinhibitory molecules, including cytotoxic T lymphocyte-associated antigen-4 (CTLA-4). Using these criteria, we detected in healthy individuals anergic T cells reactive with a skin antigen targeted in the autoimmune disease vitiligo. Collectively, our results suggest that Treg cell-mediated induction of anergy in autoimmune T cells is important for maintaining self-tolerance.

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http://dx.doi.org/10.1126/science.aaa1292DOI Listing

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