AI Article Synopsis
- A woman developed sudden bilateral ophthalmoparesis and ataxia four weeks after a bout of gastroenteritis.
- Elevated serum antibody levels against asialo-GM1 and GD1a were noted, while anti-GQ1b antibodies, usually present in Miller Fisher variant of Guillain-Barre syndrome, were absent.
- Despite thorough evaluation, no other cause for her symptoms was identified, and she improved after receiving intravenous immunoglobulin treatment.
Article Abstract
We present a woman with acute onset of bilateral ophthalmoparesis and ataxia occurring 4 weeks after gastroenteritis. Serum antibody titers against asialo-GM1 and GD1a, typically associated with inflammatory axonal neuropathies, were elevated but titers against anti-GQ1b, the most commonly found antibody found in the Miller Fisher variant of Guillain-Barre syndrome were not. No other etiology for ophthalmoparesis was found despite extensive patient evaluation. Intravenous immunoglobulin was administered, and the patient gradually improved over subsequent months. This case is unique for its antiganglioside antibody profile associated with Miller Fisher syndrome.
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| http://dx.doi.org/10.1097/WNO.0000000000000156 | DOI Listing |
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