Background: We present a case report of a 3-year-old girl who sustained a severe open fracture dislocation of her talus with complete loss of full-thickness articular cartilage and subchondral bone over 80% of the talar dome. At presentation there was an extensive soft tissue defect including absent anterior joint capsule. She required a free anterolateral thigh flap to reconstruct this defect. The talar dome defect was treated with a cell-free chondroinductive implant made of resorbable polyglycolic acid felt and hyaluronic acid. This was the first use of such an implant in the United Kingdom and the first use in a child anywhere in the world.
Methods: The case has been followed prospectively for 3 years.
Results: At 3 years postoperative, the patient underwent thinning of the anterolateral thigh flap and trimming of an anterior tibial overgrowth, which was causing impingement. At surgery the talar dome cartilage looked pristine, with a line representing the tidemark between the original cartilage and the new formed. Biopsies were taken and histopathology performed.
Conclusions: This was a rare and difficult case that has achieved an excellent outcome at this follow-up stage.
Level Of Evidence: Level V.
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http://dx.doi.org/10.1097/BPO.0000000000000198 | DOI Listing |
Foot Ankle Int
January 2025
Orthopaedic Surgery, Cedars-Sinai Medical Center, Los Angeles, CA, USA.
Arthrosc Tech
November 2024
Orthopaedic Department, Faculty of Medicine in Assiut, Al-Azhar University, Cairo, Egypt.
Osteochondral lesions of the talus are chondral lesions affecting the subchondral bone mostly due to acute ankle trauma, including either sprains or fractures. After failure of conservative treatment, operative treatment is necessary, with different surgical techniques described in the literature. We describe a single-step osteochondral autograft transfer to access the medial talar dome lesion that avoids the need for a medial malleolar osteotomy and therefore eliminates morbidity while reducing operative time.
View Article and Find Full Text PDFActa Radiol
November 2024
Radiology Department, Istanbul University, Istanbul Medical Faculty, Istanbul, Turkey.
Background: The etiology of medial-sided talar osteochondral lesions (OCLs) remains insufficiently understood.
Purpose: To identify anatomical risk factors contributing to the development of unilateral or bilateral OCL of the talus on the medial side, utilizing morphological parameters derived from magnetic resonance imaging (MRI).
Material And Methods: In this retrospective study, 24 ankle MRI scans from 12 patients exhibiting bilateral OCLs of the talar dome on the medial side, 24 ankle MRIs from 24 patients with unilateral medial-sided OCLs, and 24 healthy controls matched for age, sex, and side within each group were analyzed.
Am J Sports Med
December 2024
Linda and Mitch Hart Center for Regenerative and Personalized Medicine, Steadman Philippon Research Institute, Vail, Colorado, USA.
Background: Microfracture is one surgical treatment strategy for osteochondral lesions of the talus (OLTs) but results in fibrocartilage repair tissue, which has inferior mechanical properties to native hyaline cartilage. Biological regulation of microfracture has been suggested to improve the quality of cartilage repair in patients.
Purpose: To determine if administration of losartan, fisetin, or losartan and fisetin combined can enhance microfracture-mediated cartilage repair of OLTs in a rabbit model.
Orthop J Sports Med
October 2024
Department of Orthopedic Surgery, College of Medicine, Yeungnam University, Daegu, Republic of Korea.
Background: The pathomechanism of anterolateral ankle impingement (ALAI) due to the distal fascicle of the anterior inferior tibiofibular ligament (DF-AITFL) has not been fully elucidated. In addition, because of its rarity, no definitive diagnostic criteria have been established for ALAI due to DF-AITFL.
Purpose: To document the symptom characteristics and magnetic resonance imaging (MRI) and and arthroscopic findings as well as postoperative clinical outcomes of ALAI due to DF-AITFL.
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