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A rare congenital anomaly of the duodenal shape incidentally found at duodenopancreatectomy: A case report and literature review.
Int J Surg Case Rep
August 2021
Anatomy and Surgical Training Department, Hue University of Medicine and Pharmacy, Hue University, Hue City, Viet Nam. Electronic address:
Introduction: An anomaly of the duodenal shape is one of the rare congenital anomalies and remains poorly known in many previous studies and the literature. The duodenum is formed by developing the terminal foregut and proximal midgut through four stages in the embryonic period. According to the anatomy, the duodenum is typically described as C-shaped, U-shaped, or even horseshoe-shaped.
View Article and Find Full Text PDFHorseshoe-shaped pancreas.
JOP
January 2014
Clinical Islet Laboratory and Clinical Islet Transplant Program, University of Alberta. Edmonton, Alberta, Canada.
The reduced canine pancreas to study the effects of intra-operative radiotherapy.
Eur Surg Res
November 1993
Department of Surgical Oncology, Groningen University Hospital, The Netherlands.
A canine model is described to study the tolerance of the pancreas to intra-operative radiotherapy (IORT). The canine pancreas is a horseshoe-shaped organ. To create a homogeneous delivery of IORT to the whole pancreas surgical manipulation is necessary which may induce pancreatitis.
View Article and Find Full Text PDFHorseshoe anomaly of the pancreas.
Pancreas
August 1992
Third Department of Internal Medicine, Faculty of Medicine, Kyushu University, Fukuoka, Japan.
A 72-year-old man with recurrent pancreatitis and a horseshoe-shaped anomaly of the pancreas is described. The diagnosis was made by endoscopic retrograde cholangiopancreatography (ERCP) and computed tomography scan; laparotomy was confirmatory. The abnormal duct branched to the lower left from an enlarged Santorini's duct; a thin Wirsung's duct was joined at its distal portion to the junction of the abnormal duct.
View Article and Find Full Text PDFPatients with non-insulin-dependent diabetes mellitus have enlarged islets of Langerhans, an increased number of insulin-secreting beta cells, and a "horseshoe-shaped" relationship between plasma insulin and glucose levels. To some extent, beta-cell dysfunction in patients with type II diabetes is reversible with appropriate hypoglycemic therapy. Defects in the glucagon-secreting alpha cells in patients with non-insulin-dependent diabetes mellitus include basal hyperglucagonemia, an exaggerated glucagon response to aminogenic stimuli, and hyposuppressibility of glucagon by hyperglycemia.
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