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In this editorial, I outline two key changes to the submission guidelines, and I present my vision as the new editor for Psychology and Aging, the premier outlet for psychological research on aging and adult lifespan development. To enhance the impact of research published in the journal, my editorial team and I will accept articles that make strong theoretical contributions, are methodologically rigorous and transparent, use open science practices, contribute cumulative knowledge to the field, and have important practical implications. We will continue to publish high-quality empirical articles, systematic reviews, and meta-analyses, as well as theory development and methodological articles from all areas of psychology and related disciplines that focus on basic principles of aging and adult lifespan development or that investigate these principles in applied settings.

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Inaugural editorial.

J Pers Soc Psychol

January 2025

Department of Psychology, University at Buffalo, The State University of New York.

In this editorial, the author says that she is honored and excited to be entrusted with the responsibility of serving as editor of the Interpersonal Relations and Group Processes section of the Journal of Personality and Social Psychology. Her team is actively working to increase submissions, increase acceptances, and make the articles we ultimately publish more accessible, widening readership. She presents her team's submission and review guidelines.

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Adenine base editor corrected ADPKD point mutations in hiPSCs and kidney organoids.

Adv Biotechnol (Singap)

June 2024

MOE Key Laboratory of Gene Function and Regulation, State Key Laboratory of Biocontrol, School of Life Sciences, Sun Yat-Sen University, Guangzhou, Guangdong, 510275, China.

Autosomal dominant polycystic kidney disease (ADPKD) is a dominant genetic disorder caused primarily by mutations in the PKD1 gene, resulting in the formation of numerous cysts and eventually kidney failure. However, there are currently no gene therapy studies aimed at correcting PKD1 gene mutations. In this study, we identified two mutation sites associated with ADPKD, c.

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