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Transient acidosis during early reperfusion attenuates myocardium ischemia reperfusion injury via PI3k-Akt-eNOS signaling pathway. | LitMetric
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Transient acidosis during early reperfusion attenuates myocardium ischemia reperfusion injury via PI3k-Akt-eNOS signaling pathway.

Xin Qiao Jinjin Xu Qing-Jun Yang Yun Du Shaoqing Lei Zhi-Hong Liu Xinwei Liu Huimin Liu

Oxid Med Cell Longev

Department of Anesthesiology, Chongqing Zhoushan Hospital, Chongqing 404100, China ; Department of Anesthesiology, The First Affiliated Hospital of Chongqing Medical University, Chongqing 404100, China.

Published: July 2014

AI Article Synopsis

  • The study found that transient acidosis during reperfusion helps protect the heart from damage after a lack of blood flow (ischemia) in isolated rat hearts.
  • This protective effect is linked to the activation of a specific cellular signaling pathway called the PI3K-Akt-eNOS pathway.
  • Overall, the research suggests that manipulating pH levels during reperfusion could be a potential strategy for improving heart recovery after ischemic events.

Article Abstract

In this paper, we concluded that transient acidosis reperfusion conferred cardioprotection against myocardial ischemia reperfusion injury in isolated rat hearts through activating PI3K-Akt-eNOS pathway.

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 Source
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3839119PMC
http://dx.doi.org/10.1155/2013/126083DOI Listing

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