We report on the development of a novel shared touch-panel apparatus for examining a diverse range of topics in great ape social cognition and interaction. Our apparatus-named the Arena System-is composed of a single multitouch monitor that spans across two separate testing booths, so that individuals situated in each booth have tactile access to half of the monitor and visual access to the whole monitor. Additional components of the system include a smart-film barrier able to restrict visual access between the booths, as well as two automated feeding devices that dispense food rewards to the subjects. The touch-panel, smart-film, and feeders are controlled by a PC that is also responsible for running the experimental tasks. We present data from a pilot behavioral game theory study with two chimpanzees in order to illustrate the efficacy of our method, and we suggest applications for a range of topics including animal social learning, coordination, and behavioral economics. The system enables fully automated experimental procedures, which means that no human participation is needed to run the tasks. The novel use of a touch-panel in a social setting allows for a finer degree of data resolution than do the traditional experimental apparatuses used in prior studies on great ape social interaction.
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http://dx.doi.org/10.3758/s13428-013-0418-y | DOI Listing |
Genes Dev
December 2024
Barbara Davis Center for Diabetes, University of Colorado Anschutz Medical Campus, Aurora, Colorado 80045, USA
Transcription factors (TFs) are indispensable for maintaining cell identity through regulating cell-specific gene expression. Distinct cell identities derived from a common progenitor are frequently perpetuated by shared TFs, yet the mechanisms that enable these TFs to regulate cell-specific targets are poorly characterized. We report that the TF NKX2.
View Article and Find Full Text PDFHum Mol Genet
January 2025
Department of Ophthalmology, Baylor College of Medicine, 6565 Fannin St, NC205, Houston, TX 77030 United States.
Human diseases with similar phenotypes can be interconnected through shared biological pathways, genes, or molecular mechanisms. Inherited retinal diseases (IRDs) cause photoreceptor dysfunction due to mutations in approximately 300 genes, affecting visual transduction, photoreceptor morphogenesis, and transcription factors, suggesting common pathobiological mechanisms. This study examined the functional relationship between known IRDs genes by integrating binding sites and gene expression data from the key photoreceptor transcription factors (TFs), Crx and Nrl.
View Article and Find Full Text PDFSensors (Basel)
January 2025
Faculty of Applied Sciences, Macao Polytechnic University, Macao SAR 999078, China.
Visible-infrared person re-identification (VI-ReID) is a challenging cross-modality retrieval task to match a person across different spectral camera views. Most existing works focus on learning shared feature representations from the final embedding space of advanced networks to alleviate modality differences between visible and infrared images. However, exclusively relying on high-level semantic information from the network's final layers can restrict shared feature representations and overlook the benefits of low-level details.
View Article and Find Full Text PDFSensors (Basel)
December 2024
Institute for Production Technology and Systems (IPTS), Leuphana Universität Lüneburg, 21335 Lüneburg, Germany.
An intelligent transportation system (ITS) offers commercial and personal movement through the smart city (SC) communication paradigms with hassle-free information sharing. ITS designs and architectures have improved via information and communication technologies in recent years. The information shared through the communication medium in SCs is exposed to adversary risk, resulting in privacy issues.
View Article and Find Full Text PDFInt J Mol Sci
December 2024
Institute of Neurology, Department of Medical and Surgical Sciences, University Magna Graecia, 88100 Catanzaro, Italy.
Pathogenic variants are associated with neonatal epilepsies, ranging from self-limited neonatal epilepsy to -developmental and epileptic encephalopathy (DEE). In this study, next-generation sequencing was performed, applying a panel of 142 epilepsy genes on three unrelated individuals and affected family members, showing a wide variability in the epileptic spectrum. The genetic analysis revealed two likely pathogenic missense variants (c.
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