Objective: To examine correlates of compensatory weight control behaviors among women in transition between adolescence and adulthood.
Participants: The authors recruited a sample of undergraduate women (N = 759) at a large northwestern university during the 2009-2010 academic year.
Methods: Logistic regression was used to assess relations among childhood abuse, psychosocial functioning, adult dating relationship factors, and women's endorsement of compensatory weight control behaviors.
Results: The final model reliably distinguished between participants who endorsed versus denied use of compensatory behaviors (χ(2)[5, N = 747] = 36.37, p < .001), with global psychosocial functioning and relationship avoidance accounting for the most variance.
Conclusions: These findings illustrate the importance of considering childhood abuse histories and adult relationships while assessing young women's compensatory weight control behaviors.
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http://dx.doi.org/10.1080/07448481.2013.833515 | DOI Listing |
Soc Sci Med
January 2025
Department of Epidemiology, University of Michigan School of Public Health, 1415 Washington Heights, Ann Arbor, MI, 48109-2029, USA.
Persistent racial disparities in low birth weight (LBW) in the United States may be better understood through the adoption of a life course perspective that considers differential exposure and vulnerability of Black and White women to socioeconomic position across generations. Using a multigenerational dataset of singleton birth certificates from South Carolina from 1989 to 2020 linked along the maternal line, we constructed intergenerational social mobility trajectories of grandmaternal and maternal education and compared unadjusted and adjusted associations between trajectories and LBW among Black and White women. We found that White women were more likely to be upwardly mobile, and Black women to be downwardly mobile.
View Article and Find Full Text PDFJ Mol Neurosci
January 2025
Department of Biophysics, School of Life Sciences, Xuzhou Medical University, Xuzhou, 221004, Jiangsu, China.
Alzheimer's disease (AD), a prevalent neurodegenerative disorder, is characterized by mitochondrial dysfunction and immune dysregulation. This study is aimed at developing a risk prediction model for AD by integrating multi-omics data and exploring the interplay between mitochondrial energy metabolism-related genes (MEMRGs) and immune cell dynamics. We integrated four GEO datasets (GSE132903, GSE29378, GSE33000, GSE5281) for differential gene expression analysis, functional enrichment, and weighted gene co-expression network analysis (WGCNA).
View Article and Find Full Text PDFJ Dairy Sci
January 2025
Department of Animal Science, Penn State University, University Park, PA, USA. Electronic address:
This observational study evaluated the relationship between lung consolidation observed at weaning and calf ADG, and the association of pathogen shedding at weaning on ADG in beef × dairy calves up to 238 d. Beef × Holstein calves (n = 143) were sourced from 2 dairies. Calves were managed in 3 cohorts and fed milk replacer and calf starter preweaning.
View Article and Find Full Text PDFJ Plast Reconstr Aesthet Surg
December 2024
Department of Hand and Plastic Surgery, Thurgau Hospital Group, Frauenfeld, Switzerland; Division of Hand, Plastic and Aesthetic Surgery University Hospital, LMU Munich, Germany.
The distance between the mid-clavicle and nipple (MCN) is crucial in planning reduction mammoplasties. MCN has been shown to be superior in achieving nipple-areola complex symmetry. However, there is great variability in clavicle anatomy.
View Article and Find Full Text PDFSci Rep
January 2025
Department of Bioregulation, Institute for Advanced Medical Sciences, Nippon Medical School, Kawasaki, Kanagawa, Japan.
Insulin receptor substrate (IRS)-1 and IRS-2 are major molecules that transduce signals from insulin and insulin-like growth factor-I receptors. The physiological functions of these proteins have been intensively investigated in mice, while little is known in other animals. Our previous study showed that the disruption of IRS-2 impairs body growth but not glucose tolerance or insulin sensitivity in rats, which led us to hypothesize that IRS-1 plays more pivotal roles in insulin functions than IRS-2.
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