Objectives: There are limited data on peripheral nerve block (PNB) practices by residents in France, especially with after introduction of ultrasound. A survey was conducted on PNB practices by French residents.
Study Design: Multicenter prospective descriptive study.
Patients And Methods: All residents completed a survey form after each PNB procedure.
Results: A total of 394 procedures performed by 21 residents in 4 hospitals were collected. The number of procedures and average volume of local anesthetics by type of block are: axillary block 245-25mL (62%), interscalene 29-21mL (7%), supraclavicular 1-25mL (0.3%), 2-25mL infraclavicular (0.5%), femoral 65-18mL (16.5%), sciatic 52-22mL (13%). Detection was done by ultrasound only for 365 (92.6%) procedures, and neurostimulation only for 23 (5.8%) procedures. The duration of procedure was less than 5min for 200 (50.7%) procedures and more than 15min for 16 (4%) procedures. A single puncture was required for 307 (78%) procedures. The initial block success was obtained in 369 (93%) procedures. High training level residents required less time (P<0.0001), less puncture (P<0.046) and less neurostimulation use (P<0.0001) than novices, without difference in success rate and the volume of local anesthetic required.
Conclusions: The majority of procedures performed by the residents are using ultrasound. The procedure is usually fast, requiring mainly a single puncture and has a high rate of success.
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http://dx.doi.org/10.1016/j.annfar.2013.07.811 | DOI Listing |
Sci Rep
December 2024
Department of Orthopaedic Surgery, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-Ku, Tokyo, 160-8582, Japan.
Chronic complete spinal cord injury (SCI) is difficult to treat because of scar formation and cavitary lesions. While human iPS cell-derived neural stem/progenitor cell (hNS/PC) therapy shows promise, its efficacy is limited without the structural support needed to address cavitary lesions. Our study investigated a combined approach involving surgical scar resection, decellularized extracellular matrix (dECM) hydrogel as a scaffold, and hNS/PC transplantation.
View Article and Find Full Text PDFJ Med Case Rep
December 2024
Division of Infectious Diseases, Denver Health Medical Center, Denver, CO, USA.
Background: Leprosy (Hansen's disease) is an infectious disease most common in resource-limited countries caused by the acid-fast bacilli Mycobacterium leprae and Mycobacterium lepromatosis that frequently affects the skin and peripheral nerves. Prompt diagnosis and treatment with multidrug therapy is crucial to reduce disease transmission and sequelae, which include nerve function impairment, ocular injury, and stigmatizing physical deformities. Traditional treatment of multibacillary leprosy consists of 12-24 months of multidrug therapy with dapsone, rifampin, and clofazimine.
View Article and Find Full Text PDFIntroduction: Available therapies for peripheral nerve injury (PNI) include surgical and non-surgical treatments. Surgical treatment includes neurorrhaphy, grafting (allografts and autografts) and tissue-engineered grafting (artificial nerve guide conduits), while non-surgical treatment methods include electrical stimulation, magnetic stimulation, laser phototherapy and administration of nerve growth factors. However, the treatments currently available to best manage the different PNI manifestations remain undetermined.
View Article and Find Full Text PDFJ Oral Maxillofac Surg
December 2024
PhD Adjunct Professor, in Oral and Maxillofacial Surgery, School of Dentistry, University of Pernambuco - UPE, Recife, Pernambuco, Brazil. Electronic address:
Background: Fluoxetine, a serotonin reuptake inhibitor antidepressant, raises extracellular serotonin levels and promotes angiogenesis and neurogenesis. Numerous animal models have shown its beneficial effects on recovery from peripheral nerve injury.
Purpose: The primary objective of this study was to analyze the influence of fluoxetine on the sensory-motor function recovery of the sciatic nerve in Wistar rats after axonotmesis.
Neurobiol Dis
December 2024
Department of Neurology, University Hospital of Wuerzburg, Germany. Electronic address:
DYT-THAP1 dystonia is a monogenetic form of dystonia, a movement disorder characterized by the involuntary co-contraction of agonistic and antagonistic muscles. The disease is caused by mutations in the THAP1 gene, although the precise mechanisms by which these mutations contribute to the pathophysiology of dystonia remain unclear. The incomplete penetrance of DYT-THAP1 dystonia, estimated at 40 to 60 %, suggests that an environmental trigger may be required for the manifestation of the disease in genetically predisposed individuals.
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