Multiple Calcifying Hyperplastic Dental Follicle (MCHDF): A Case Report.

J Dent Res Dent Clin Dent Prospects

Department of Oral and Maxillofacial Pathology, Faculty of Dentistry, Hamadan University of Medical Sciences, Hamadan, Iran.

Published: October 2013

Evaluation of enlarged follicles with unerupted teeth is always important because some changes may occur. One of the ex-tremely rare conditions seen in dental follicles is multiple calcifying hyperplastic dental follicle. We report a case of multi-ple calcifying hyperplastic dental follicle. Radiographs showed that mandibular third molars had a pericoronal radiolucent zone delineated by a well-defined and sclerotic border. Microscopic examination revealed a combination of fibrous connec-tive and small calcifications. Multiple calcifying hyperplastic dental follicle is a rare condition and its correct diagnosis is necessary to apply appropriate treatment.

Download full-text PDF

Source
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3779378PMC
http://dx.doi.org/10.5681/joddd.2013.028DOI Listing

Publication Analysis

Top Keywords

calcifying hyperplastic
16
hyperplastic dental
16
dental follicle
16
multiple calcifying
12
dental
5
hyperplastic
4
follicle
4
follicle mchdf
4
mchdf case
4
case report
4

Similar Publications

Background: Coronary artery disease is the most common form of cardiovascular disease. It is caused by excess plaque along the arterial wall, blocking blood flow to the heart (stenosis). A percutaneous coronary intervention widens the arterial wall with the inflation of a balloon inside the lesion area and leaves behind a metal stent to prevent re-narrowing of the artery (restenosis).

View Article and Find Full Text PDF

Background: Despite improvement in devices, in-stent restenosis remains a frequent and challenging complication of percutaneous coronary interventions.

Methods And Results: The RESTO (Morphological Parameters of In-Stent Restenosis Assessed and Identified by OCT [Optical Coherence Tomography]; study NCT04268875) was a prospective multicenter registry including patients presenting with coronary syndromes related to in-stent restenosis. All patients underwent preintervention OCT analysis, which led to analysis of in-stent restenosis phenotype, number of strut layers, and presence of stent underexpansion.

View Article and Find Full Text PDF
Article Synopsis
  • Vascular calcification (VC) involves calcium buildup in the aorta, increasing cardiovascular risks, and the study investigates the role of nesfatin-1 in this process.
  • Higher levels of nesfatin-1 were found in calcified vascular smooth muscle cells (VSMCs) and patients with coronary calcification, with experiments showing it regulates VC by promoting osteogenic transformation of VSMCs.
  • The study discovered that nesfatin-1 enhances BMP-2 signaling by inhibiting SYTL4, leading to changes that promote transcription of OPN, and identified compounds like Curculigoside and Chebulagic acid that reduce VC by interacting with nesfatin-1.
View Article and Find Full Text PDF

Background: The presentation of mineral bone disorder (MBD) in individuals with chronic kidney disease (CKD) may vary. Consequently, physicians should be capable of recognising this condition when there is a suspicion of its existence. This case report will describe a calcified maxilla tumour as a manifestation of CKD-MBD.

View Article and Find Full Text PDF

Self-expandable stents manufactured from nitinol alloys are commonly utilized alongside traditional balloon-expandable stents to provide scaffolding to stenosed arteries. However, a significant limitation hampering stent efficacy is restenosis, triggered by neointimal hyperplasia and resulting in the loss of gain in lumen size, post-intervention. In this study, a nonlinear finite element model was developed to simulate stent crimping and expansion and its interaction with the surrounding vessel in the presence of a plaque.

View Article and Find Full Text PDF

Want AI Summaries of new PubMed Abstracts delivered to your In-box?

Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!