We describe the scaling up of an evidence-informed model of care, Managing and Adapting Practice (MAP) in Los Angeles County, California. MAP complemented an array of evidence-based programs selected by the county as part of a large system reform effort designed to improve care for children and adolescents. In addition, we discuss the MAP model for training therapists and present data both on how the training model performed and on the outcomes of youths treated by therapists trained in MAP. We examined the success of two different training pathways for MAP therapists: (a) national training model and (b) MAP agency supervisor model (i.e., train the trainer). We also examined utilization of MAP and outcomes of clients served by MAP. Both the national training and MAP agency supervisor model were successful in producing MAP therapists in a timely fashion and with acceptable competency scores. Furthermore, a large number of clients were receiving MAP services. Finally, outcomes for youth treated with MAP were strong, with effect sizes ranging from .59 to .80 on the Youth Outcome Questionnaire. These data support the notion that scaling up a mental health services approach in a system can be achieved through a strong and broad partnership among relevant stakeholders, can involve a train-the-trainer model, and can result in strong outcomes for clients.
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http://dx.doi.org/10.1080/15374416.2013.833098 | DOI Listing |
BMC Public Health
January 2025
National Institute for Health Research (NIHR) School for Public Health Research (SPHR), Newcastle, UK.
Background: In England, 23% of children aged 11 start their teenage years living with obesity. An adolescent living with obesity is five times more likely to live with obesity in adult life. There is limited research and policy incorporating adolescents' views on how they experience the commercial determinants of dietary behaviour and obesity, which misses an opportunity to improve services and policies that aim to influence the prevalence of childhood obesity.
View Article and Find Full Text PDFACS Nano
January 2025
Peter Grünberg Institut (PGI-9), Forschungszentrum Jülich, 52425 Jülich, Germany.
The combination of an ordinary s-type superconductor with three-dimensional topological insulators creates a promising platform for fault-tolerant topological quantum computing circuits based on Majorana braiding. The backbone of the braiding mechanism are three-terminal Josephson junctions. It is crucial to understand the transport in these devices for further use in quantum computing applications.
View Article and Find Full Text PDFSci Rep
January 2025
Department of Mathematics, College of Science, King Khalid, University, Abha, 61413, Saudi Arabia.
Algebraic structures play a vital role in securing important data. These structures are utilized to construct the non-linear components of block ciphers. Since constructing non-linear components through algebraic structures is crucial for the confusion aspects of encryption schemes, relying solely on these structures can result in limited key spaces.
View Article and Find Full Text PDFNat Metab
January 2025
Westlake Center for Intelligent Proteomics, Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, China.
The blood proteome contains biomarkers of ageing and age-associated diseases, but such markers are rarely validated longitudinally. Here we map the longitudinal proteome in 7,565 serum samples from a cohort of 3,796 middle-aged and elderly adults across three time points over a 9-year follow-up period. We pinpoint 86 ageing-related proteins that exhibit signatures associated with 32 clinical traits and the incidence of 14 major ageing-related chronic diseases.
View Article and Find Full Text PDFCell Death Dis
January 2025
Division of Hematology and Oncology, Department of Pediatrics, Penn State College of Medicine, Hershey, PA, USA.
The association of necrosis in tumors with poor prognosis implies a potential tumor-promoting role. However, the mechanisms underlying cell death in this context and how damaged tissue contributes to tumor progression remain unclear. Here, we identified p38 mitogen-activated protein kinases (p38 MAPK, a.
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