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Drug-induced lupus erythematosus (DILE) is an autoimmune reaction that results in symptoms of polyarthralgia, fever, and cutaneous lesions and other manifestations. Several drugs have been documented to cause this disease, including procainamide, isoniazid, methyldopa, penicillamine, and hydralazine. Systemic lupus erythematosus (SLE) manifestations often occur after the patient has been taking the drug without complications for months to years.
View Article and Find Full Text PDFUnraveling the Growth Mechanism of Chiral Inorganic Nanocrystals via High-Resolution Electron Microscopy.
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School of Physical Science and Technology & Shanghai Key Laboratory of High-Resolution Electron Microscopy, ShanghaiTech University, Shanghai 201210, China.
Chiral inorganic nanomaterials have attracted broad interest due to their intriguing chirality-dependent performances. However, there is a lack of experimental studies and atomic-level evidence on their growth mechanism. Herein, high-crystalline chiral tellurium nanowires were synthesized in an alkali solution by using tellurium oxide as an inorganic source and hydrazine hydrate as a reductant.
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Department of Pharmacy, Houston Methodist Hospital, Houston, TX, USA.
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View Article and Find Full Text PDFMechanisms of delta opioid receptor inhibition of parallel fibers-purkinje cell synaptic transmission in the mouse cerebellar cortex.
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Department of Physiology and Pathophysiology, College of Medicine, Yanbian University, Yanji City, Jilin Province, 133002, China. Electronic address:
Delta opioid receptors (DORs) are widely expressed throughout the central nervous system, including the cerebellum, where they play a regulatory role in neurogenesis. In the cerebellar cortex, Purkinje cells (PCs), the sole output neurons, receive glutamatergic synaptic input from parallel fibers (PFs)-the axonal extensions of granule cells-forming PF-PC synapses. However, the precise distribution of DORs within these synapses and their impact on synaptic transmission remain unclear.
View Article and Find Full Text PDFPEGylation of indium phosphide quantum dots prevents quantum dot mediated platelet activation.
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Biomedical Institute for Multimorbidity, Hull York Medical School, University of Hull, Hull, HU6 7RX, UK.
Article Synopsis
- Quantum dots (QDs) are small semiconductor particles that could improve biomedical imaging and drug delivery, with Indium phosphide QDs covered by zinc sulphide being a more biocompatible option.
- This study reveals that PEGylating these QDs significantly reduces platelet activation and aggregation, which is important to prevent excessive blood clotting.
- By decreasing the interaction between QDs and platelets, PEGylation enhances the safety and effectiveness of QDs for use in medical applications.
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