Primary Objective: The purpose of this study was to determine the prevalence of potential mild brain injury incidents accompanied by behavioural changes in a non-clinical population of elementary school-aged children. The researchers also examined data regarding the occurrence of possible brain injuries among regular vs special education students.
Research Design: Survey study.
Methods And Procedures: The researchers distributed questionnaires through which parents or guardians of 692 first-to-fifth grade students provided information about possible brain injury incidents and associated behavioural changes.
Main Outcomes And Results: Findings suggest that as many as one-third of children experience one or more incidents potentially resulting in brain injury before completing elementary school. Despite this, only 7.66% of the sample received positive screen results because of behavioural changes associated with a potential incident. None of the 53 children with positive screens were receiving special education services through the traumatic brain injury category of IDEA at the time of the screening. Positive screens were more common among students verified for special education than those enrolled in regular education. Specifically, 25.40% of positive screen results were for children identified with a disability warranting special education services; in contrast, only 5.90% of regular education students received positive screen results.
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http://dx.doi.org/10.3109/02699052.2013.823657 | DOI Listing |
Appl Neuropsychol Child
January 2025
Department of Psychology and Neuroscience Center, Brigham Young University, Provo, USA.
Chronic stage neuropsychological assessments of children with severe TBI typically center around a referral question and focus on assessing cognitive, behavioral, and emotional functioning, making differential diagnoses, and planning treatment. When severe TBI-related neurological deficits are subtle and fall outside commonly assessed behavioral indicators, as can happen with theory of mind and social information processing, they can go unobserved and subsequently fail to be assessed. Additionally, should chronic stage cognitive, behavioral, and emotional assessment findings fall within the average to above average range, a child experiencing ongoing significant unassessed severe TBI-related subtle deficits could be mistakenly judged to have "recovered" from their injury; and to be experiencing no significant ongoing residual neurological deficits.
View Article and Find Full Text PDFNeurosurg Rev
January 2025
Lab in Biotechnology and Biosignal Transduction, Department of Orthodontics, Saveetha Dental College and Hospital, Saveetha Institute of Medical and Technical Sciences (SIMATS), Saveetha University, Chennai-77, Tamil Nadu, India.
Histol Histopathol
February 2025
Department of Intensive Care Unit, Affiliated Cancer Hospital and Institute of Guangzhou Medical University, Guangzhou, Guangdong, China.
The authors regret the paper was published with an error in Figure 3B sh-NC+HI group. The H&E image in 3B sh-NC+HI group should be corrected as follows. This correction has no influence on the conclusion and the main text of the article.
View Article and Find Full Text PDFFront Aging Neurosci
January 2025
School of Medicine, Yunnan University, Kunming, China.
Background: Traumatic brain injury (TBI) can generally be divided into focal damage and diffuse damage, and neonate Hypoxia-Ischemia Brain Damage (nHIBD) is one of the causes of diffuse damage. Patients with nHIBD are at an increased risk of developing Alzheimer's disease (AD). However, the shared pathogenesis of patients affected with both neurological disorders has not been fully elucidated.
View Article and Find Full Text PDFBiomater Transl
November 2024
Key Laboratory of Spine and Spinal Cord Injury Repair and Regeneration of Ministry of Education, Department of Orthopedics, Tongji Hospital affiliated to Tongji University, School of Life Science and Technology, Tongji University, Shanghai, China.
Stem cell-derived spinal cord organoids (SCOs) have revolutionised the study of spinal cord development and disease mechanisms, offering a three-dimensional model that recapitulates the complexity of native tissue. This review synthesises recent advancements in SCO technology, highlighting their role in modelling spinal cord morphogenesis and their application in neurodegenerative disease research. We discuss the methodological breakthroughs in inducing regional specification and cellular diversity within SCOs, which have enhanced their predictive ability for drug screening and their relevance in mimicking pathological conditions such as neurodegenerative diseases and neuromuscular disorders.
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