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Matrix metalloproteinase-1 (MMP-1) and (MMP-8) gene polymorphisms promote increase and remodeling of the collagen III and V in posterior tibial tendinopathy.
Histol Histopathol
September 2018
Department of Pathology, University of São Paulo, São Paulo, Brazil.
Posterior tibial tendinopathy (PTT) can lead to acquired flatfoot in adults. Many patients develop PTT without any identifiable risk factors. Molecular changes in extracellular matrix (ECM) and matrix metalloproteinase (MMP) polymorphism may influence the risk of developing PTT.
View Article and Find Full Text PDFLow-intensity training provokes adaptive extracellular matrix turnover of a muscular dystrophy model.
J Exerc Rehabil
December 2017
Faculty of Medicine FAMED, Universidade Federal dos Vales do Jequitinhonha e Mucuri (UFVJM), Diamantina, Brazil.
Recommendations of therapeutic exercise in Duchenne muscular dystrophy are still controversial. The hypothesis that a low-intensity training (LIT) protocol leads to muscle adaptations on mdx mice model was tested. Dystrophic male mice with 8 weeks old were separated in exercised (mdxE, n= 8) and sedentary (mdxC, n= 8) groups.
View Article and Find Full Text PDFDystrophic spinal deformities in a neurofibromatosis type 1 murine model.
PLoS One
March 2016
Department of Anatomy and Cell Biology, Indiana University School of Medicine, Indianapolis, Indiana, United States of America; Herman B. Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, Indiana, United States of America; Department of Pediatrics, Indiana University School of Medicine, Indianapolis, Indiana, United States of America.
Despite the high prevalence and significant morbidity of spinal anomalies in neurofibromatosis type 1 (NF1), the pathogenesis of these defects remains largely unknown. Here, we present two murine models: Nf1flox/-;PeriCre and Nf1flox/-;Col.2.
View Article and Find Full Text PDFHistomorphometry of dystrophic fat in a patient suffering Madelung's disease.
J Plast Reconstr Aesthet Surg
January 2014
Department of Plastic and Reconstructive, CTO-AOUC, University of Florence, Italy.
The effects of glucocorticoid and voluntary exercise treatment on the development of thoracolumbar kyphosis in dystrophin-deficient mice.
PLoS Curr
October 2012
Arizona College of Osteopathic Medicine, Midwestern University.
The development of spinal curvature deformities is a hallmark of muscular dystrophy. While glucocorticoid treatment has been shown to prolong muscle function in dystrophic mice, its effects on the development of dystrophinopathic spinal deformation are poorly understood. In this study, we test the effects of glucocorticoid treatment on the onset of thoracolumbar kyphosis in the dystrophin-deficient mdx mouse using voluntary running exercise to exacerbate muscle fibrosis.
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