Download full-text PDF |
Source |
|---|---|
| http://dx.doi.org/10.1007/s00408-013-9496-y | DOI Listing |
Publication Analysis
Top Keywords
Similar Publications
Glucagon-like peptide (GLP)-1 regulation of lipid and lipoprotein metabolism.
Med Rev (2021)
August 2024
Molecular Medicine, Research Institute, The Hospital for Sick Children, University of Toronto, Toronto, ON, Canada.
Metabolic health is highly dependent on intestinal and hepatic handling of dietary and endogenous lipids and lipoproteins. Disorders of lipid and lipoprotein metabolism are commonly observed in patients with insulin resistant states such as obesity, metabolic syndrome, and type 2 diabetes. Evidence from both animal models and human studies indicates that a major underlying factor in metabolic or diabetic dyslipidemia is the overproduction of hepatic and intestinal apolipoprotein (apo)B-containing lipoprotein particles.
View Article and Find Full Text PDFIdentification of vagal afferent nerve endings in the mouse colon and their spatial relationship with enterochromaffin cells.
Cell Tissue Res
June 2024
Visceral Neurophysiology Laboratory, Flinders Health and Medical Research Institute & College of Medicine and Public Health, Flinders University of South Australia, GPO Box 2100, Bedford Park, Adelaide, South Australia, 5042, Australia.
Understanding how the gut communicates with the brain, via sensory nerves, is of significant interest to medical science. Enteroendocrine cells (EEC) that line the mucosa of the gastrointestinal tract release neurochemicals, including the largest quantity of 5-hydroxytryptamine (5-HT). How the release of substances, like 5-HT, from enterochromaffin (EC) cells activates vagal afferent nerve endings is unresolved.
View Article and Find Full Text PDFSelective KCNQ2/3 Potassium Channel Opener ICA-069673 Inhibits Excitability in Mouse Vagal Sensory Neurons.
J Pharmacol Exp Ther
March 2024
Division of Allergy and Clinical Immunology, Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, Maryland.
Heightened excitability of vagal sensory neurons in inflammatory visceral diseases contributes to unproductive and difficult-to-treat neuronally based symptoms such as visceral pain and dysfunction. Identification of targets and modulators capable of regulating the excitability of vagal sensory neurons may lead to novel therapeutic options. genes encode K7.
View Article and Find Full Text PDFInvestigating seed bank potential of crustose coralline algae using DNA metabarcoding.
J Phycol
February 2024
School of Biological Sciences, University of Auckland, Auckland, New Zealand.
To examine the potential for the autogenic ecosystem engineers, crustose coralline algae (CCA), to serve as seed banks or refugia for life stages of other species, it is critical to develop sampling protocols that reflect the diversity of life present. In this pilot study on two shallow water species of CCA collected from Raoul Island (Kermadec Islands; Rangitāhua) New Zealand, we investigated two preservation methods (ethanol vs. silica gel), sampled inner and outer regions of the crusts, and used DNA metabarcoding and seven genes/gene regions (16S rRNA, 18S rRNA, 23S rRNA, cox1, rbcL, and tufA genes and the ITS rRNA region) to develop a protocol for taxa identification.
View Article and Find Full Text PDFTaxonomy and phylogeny of brown-rot corticioid fungi in China: and spp. nov.
Front Microbiol
March 2023
School of Ecology and Nature Conservation, Beijing Forestry University, Beijing, China.
Brown-rot fungi account for a small portion of the wood-decaying fungi. There are a few corticioid genera causing brown rot of wood, and their species diversity is still under investigated and studied, especially in subtropical and tropical areas. Two new brown-rot corticioid fungi, and were found during the investigation of corticioid fungi in China.
View Article and Find Full Text PDFWant AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!