Background: In this case report, we describe an unusual case of a patient with myoclonus only occurring during menses.
Case Report: A 41-year-old female, known to have neurological sequelae after a car accident 1 year earlier, presented with myoclonic movements of the right arm and hand only during menses. Brain magnetic resonance imaging is compatible with head trauma. Electromyography shows brief irregular bursts with a duration of about 20 ms.
Discussion: This appears to be the first description of myoclonus appearing only during menses. We suggest a cortical origin for myoclonus.
Download full-text PDF |
Source |
|---|---|
| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3636523 | PMC |
| http://dx.doi.org/10.7916/D83X85C9 | DOI Listing |
Publication Analysis
Top Keywords
Similar Publications
Meningeal Dissemination and Drop Metastasis From Glioma Presenting With Non-Epileptic Myoclonus and Minipolymyoclonus.
Neurohospitalist
October 2024
Department of Neurology, Johns Hopkins University, Baltimore, MD, USA.
We describe the case of a 36-year-old woman with a past medical history of low grade right frontal lobe glioma and focal epilepsy presenting with subacute, progressive, multifocal myoclonus and neck and back pain. Unlike her typical seizures, the myoclonus exhibited a distinct semiology, involving both positive and negative muscle jerks affecting multiple limb muscles while sparing the face. In addition, neurological examination revealed low-amplitude, arrhythmic movements of the hands and fingers, resembling minipolymyoclonus.
View Article and Find Full Text PDFSporadic Creutzfeldt-Jakob Disease: A Rare Case of Rapid Progressive Cognitive Decline with Special Reference to Magnetic Resonance Spectroscopy.
J Assoc Physicians India
November 2024
Associate Professor, Department of General Medicine, Medical College Baroda, Vadodara, Gujarat, India.
Article Synopsis
- - Creutzfeldt-Jakob disease (CJD) is a rare and fatal brain disorder that typically affects people in their 60s and manifests as rapid cognitive decline due to the accumulation of abnormal prion proteins, leading to death within a year of diagnosis.
- - Diagnosis of CJD is complicated by overlapping symptoms with other degenerative and infectious diseases, requiring a comprehensive approach that includes clinical evaluations and advanced imaging techniques.
- - A case study of a 60-year-old man with probable sporadic CJD highlights symptoms such as dementia and rigidity, as well as MRI findings that indicate severe neurodegeneration, demonstrating the need for early identification for palliative care, since there is no effective treatment.
Autoimmune glial fibrillary acidic protein (GFAP) astrocytopathy is an immune-mediated inflammatory disease of the central nervous system, the clinical phenotype of which includes meningoencephalitis, myelitis, optic neuritis, seizures, movement disorders, autonomic dysfunction among others, affecting people across all ages. Due to its recent discovery, there is a paucity of literature on this topic and an absolute lack of Indian case series. This study aims to shed light on the variable presentations of anti-GFAP astrocytopathy and review the existing literature on the topic.
View Article and Find Full Text PDFDevelopment of cefepime-induced encephalopathy in a patient with depression and rectal cancer: A case report.
Neuropsychopharmacol Rep
November 2024
National Cancer Center Hospital, Tokyo, Japan.
Article Synopsis
- Cefepime, a fourth-generation antibiotic, can cause neurotoxic side effects like encephalopathy, particularly in patients with compromised blood-brain barriers (BBB) and renal issues.
- A case study of a 79-year-old woman with depression and cancer demonstrated that after receiving cefepime, she developed confusion and other neurological symptoms without apparent brain damage or kidney dysfunction.
- The case suggests that depression and cancer may increase the risk of cefepime-induced encephalopathy, highlighting the need for doctors to monitor such patients closely to avoid misdiagnosis and ensure proper treatment.
Multimodal Assessment of the Origin of Myoclonus in Lance-Adams Syndrome.
Neurology
December 2024
From the Paris Brain Institute (G.V., E.A., P.B., V.N.), ICM, Inserm, CNRS, Sorbonne University; AP-HP (G.V., V.N.), EEG Unit, Department of Neurophysiology, Pitié-Salpêtrière Hospital; AP-HP (E.A.), Neurophysiology of Movement Disorders Unit, Department of Neurophysiology, Saint-Antoine and Pitié-Salpêtrière Hospital; AP-HP (M.A.D.R.Q., V.N.), Epilepsy Unit, Department of Neurology, Reference Center of Rare Epilepsies, ERN-EpiCare, Pitié-Salpêtrière Hospital; AP-HP (D.V.C., A.K.), Department of Nuclear Medicine, Pitié-Salpêtrière Hospital, APHP Sorbonne University; Sorbonne University (A.K.), Inserm, CNRS, Laboratoire D'Imagerie Biomédicale, LIB, Paris, France.
Background And Objectives: Lance-Adams syndrome (LAS), or chronic posthypoxic myoclonus, is a long-term disabling neurologic disorder occurring in survivors of anoxia. The cortical or subcortical origin of this myoclonus is unclear. We aimed to identify the neuroanatomical origin of myoclonus in LAS.
View Article and Find Full Text PDFWant AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!