AI Article Synopsis

  • Magnesium is considered a promising biodegradable material, and this study focused on creating forsterite-containing coatings for ZK60 magnesium alloy using a silicate electrolyte-based micro-arc oxidation (MAO) method to improve its biological properties and decrease its degradation rate.
  • The research involved testing the MAO coatings at four anodization voltages, with results showing no cytotoxicity to L929 cells and a significant reduction in hemolytic ratio compared to uncoated alloy, indicating better biocompatibility.
  • Analysis of corrosion resistance revealed that the MAO coatings effectively reduced the degradation rate of the alloy, with increased resistance observed at higher preparation voltages, leading to the proposal of a degradation model for the coated ZK60

Article Abstract

Magnesium has attracted much attention as a class of biodegradable metallic biomaterials. In this study, a silicate electrolyte-based micro-arc oxidation (MAO) treatment was adopted to prepare forsterite-containing MAO coatings on a ZK60 magnesium alloy in order to decrease the degradation rate and increase the biological property of the alloy. Four anodization voltages were chosen to prepare the MAO coatings. The cell experiment showed a cytotoxicity of grade 0 for the MAO-coated alloy to L929 cells and the hemolytic ratio was dramatically decreased for the MAO-coated alloy compared with the bare one. The corrosion resistance and the degradation behavior of the MAO-coated ZK60 alloy were studied using drop tests, electrochemical measurements and immersion tests. The results indicate that the MAO coating could effectively decrease the initial degradation rate of the alloy. The corrosion resistance of MAO coating was increased with the elevation of the preparation voltage. A degradation model for ZK60 alloy with a forsterite-containing MAO coating was proposed. Based on the model, the MAO-coated alloy experiences destruction and restoration simultaneously, and the coating fails in a peeling-off mode.

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http://dx.doi.org/10.1016/j.actbio.2012.12.016DOI Listing

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