This study assessed the asbestos exposures of airplane piston engine mechanics while performing overhaul work on a Pratt & Whitney R2800 radial engine, with tools and practices in use since the time these engines were manufactured. Approximately 40% of the bulk samples collected during this test were found to contain chrysotile. Air samples were collected during the overhaul and were analyzed by phase contrast microscopy (PCM) and transmission electron microscopy (TEM). The average worker exposure during disassembly was 0.0272f/ml (PCM) and ranged from 0.0013 to 0.1240f/ml (PCM) during an average sample collection time of 188min. The average worker exposure during reassembly was 0.0198f/ml (PCM) and ranged from 0.0055 to 0.0913f/ml (PCM) during an average sample collection time of 222min. Only one worker sample (during reassembly) was found to contain asbestos at a concentration of 0.0012f/ml (PCME). Similar results should be found in other aircraft piston engines that use metal clad and non-friable asbestos gaskets, which are the current standard in aircraft piston engines.
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http://dx.doi.org/10.1016/j.yrtph.2012.07.004 | DOI Listing |
Alcohol Alcohol
September 2009
Institute of Psychiatric Research, 791 Union Drive, Indianapolis, IN 46202-4887, USA.
Aims: The present study sought to investigate the relationship between the HPA axis reactivity to stress, the endogenous opioid system and stress-induced drinking behavior.
Methods: In the present study, 74 non-treatment-seeking alcohol-dependent subjects were tested under two mood conditions, neutral and stress, in separate testing sessions. Salivary cortisol measurements were obtained following stress induction and during the neutral control condition.
Alcohol
November 2005
Institute of Psychiatric Research, Department of Psychiatry, Indiana University School of Medicine, 791 Union Drive, Indianapolis, IN 46202-4887, USA.
Am J Respir Cell Mol Biol
October 2005
Department of Cellular and Integrative Physiology, Indiana University School of Medicine, Indianapolis, Indiana, USA.
Hypoxic proliferation of pulmonary arterial smooth muscle cells (PASMC) is mitogen dependent, but the signaling pathways mediating hypoxia-induced cell growth are not well understood. We investigated hypoxic proliferation in primary cultures from porcine pulmonary artery smooth muscle. The cells were grown in medium with or without platelet-derived growth factor (PDGF)-B, a potent smooth muscle cell mitogen.
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