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Hemochromatosis-induced bipolar disorder: a case report.
Gen Hosp Psychiatry
June 2012
NESMOS (Neurosciences, Mental Health and Sensory Functions) Department, School of Medicine and Psychology, Sapienza University, Sant'Andrea Hospital, UOC Psychiatry, Via di Grottarossa 1035-1037, 00189 Rome, Italy.
Objective: A patient presenting with Diagnostic and Statistical Manual of Mental Disorders, Fourth Edition, bipolar disorder was found to be affected by high iron hemochromatosis. This prompted us to explore the relation between bipolar disorder and iron overload.
Method: We report the case and review the peer-reviewed literature focusing on mood symptoms in patients with hemochromatosis or iron overload.
'A man can be destroyed but not defeated': Ernest Hemingway's near-death experience and declining health.
Front Neurol Neurosci
May 2010
Laboratory of Cognitive Neuroscience, Ecole Polytechnique Fédérale de Lausanne, Lausanne, Switzerland.
Ernest Hemingway is one of the most popular and widely acclaimed American writers of the 20th century. His works and life epitomize the image of the hyper-masculine hero, facing the cruelties of life with 'grace under pressure'. Most of his writings have a quasi-autobiographical quality, which allowed many commentators to draw comparisons between his personality and his art.
View Article and Find Full Text PDFStrategies for dissecting genetic-environmental interactions in neurodegenerative disorders.
Neurotoxicology
September 2006
Joseph and Kathleen Bryan Alzheimer Disease Research Center, Department of Medicine (Neurology), Duke University Medical Center, Durham, NC 27710, USA.
Complex genetic and environmental interactions contribute to abnormal aging and neurodegenerative disorders. We present information from a series of 1136 consecutive patients presenting with cognitive disorders and show possible significant contribution of toxic environmental and occupational exposures to pathological aging (21% of patients) and interactions of these exposures with common polymorphisms that affect cell injury and inflammation. Such exposures may lower age of onset to same degree as APOE4/4.
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