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Reappraisal of the electrographic manifestations of right ventricular apical pacing.
J Electrocardiol
November 2012
Florida Heart Rhythm Institute, Tampa, FL, USA.
"All that spikes is not fits", mistaking the woods for the trees: the interictal spikes--an "EEG chameleon" in the interface disorders of brain and mind: a critical review.
Clin EEG Neurosci
October 2009
Department of Neurology, Father Muller Medical College, Mangalore 575 002, Kamataka, India.
Recent research into mammalian cortical neurophysiology, after 6 decades of Berger's seminal work on electroencephalography, has shifted the older concept of interictal epileptiform activity (IEA) away from that of a mere electrographic graphoelement of relevance to diagnostic implications in epilepsy. Instead, accumulating information has stressed the neuropsychological implications, cognitive and/or behavioral consequence of these electrophysiological events, which are the phenotypic expression of aberrations of actual biophysical cellular function. We feel that this review is germane to neuropsychiatry, however, a rather neglected area of research.
View Article and Find Full Text PDFA reappraisal of pacemaker timing cycles pertaining to automatic mode switching.
J Interv Card Electrophysiol
December 2001
Department of Cardiology, AZ Damiaan Hospital, Oostende, Belgium.
Automatic mode switching algorithms of dual chamber pacemakers require fundamental changes in the operation of pacemaker timing cycles to optimize detection of supraventricular tachyarrhythmias. The timing cycles related to mode switching are basically independent of the algorithm design. Blanking periods (when the sensing amplifier is temporarily disabled) should be optimized to a relatively small fraction of the pacing cycle to enhance atrial sensing and prevent far-field sensing.
View Article and Find Full Text PDFTorsade de pointes as a complication of subarachnoid hemorrhage: a critical reappraisal.
J Electrocardiol
January 1997
Arrhythmia Center, Division of Cardiology/Department of Internal Medicine, Sinai Hospital, Detroit, Michigan, USA.
Subarachnoid hemorrhage is widely accepted as a potential cause of torsade de pointes (TdP), yet this putative etiologic relationship has never been systematically evaluated. We therefore undertook a MEDLINE search from 1966 through 1993, with relevant back referencing, and identified 20 cases of TdP in the setting of subarachnoid hemorrhage. It was impossible in any of these cases (usually because of insufficient data) to completely exclude one or more alternative explanations for TdP, including congenital long QT syndrome, hypokalemia, hypomagnesemia, or drug-induced QT prolongation.
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