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Multi-Omics Research on Angina Pectoris: A Novel Perspective.
Aging Dis
December 2024
Department of Psycho-cardiology, Beijing Anzhen Hospital, Capital Medical University, Beijing, China.
Angina pectoris (AP), a clinical syndrome characterized by paroxysmal chest pain, is caused by insufficient blood supply to the coronary arteries and sudden temporary myocardial ischemia and hypoxia. Long-term AP typically induces other cardiovascular events, including myocardial infarction and heart failure, posing a serious threat to patient safety. However, AP's complex pathological mechanisms and developmental processes introduce significant challenges in the rapid diagnosis and accurate treatment of its different subtypes, including stable angina pectoris (SAP), unstable angina pectoris (UAP), and variant angina pectoris (VAP).
View Article and Find Full Text PDFCoronary vasomotor response incidence to intracoronary acetylcholine provocation test according to the severity of insignificant coronary artery stenosis in Korean population.
Coron Artery Dis
December 2024
Department of Biomedical Laboratory Science, Honam University, Gwangju, Korea.
Background: Coronary artery spasm (CAS) is a cause of variant angina. However, the understanding of CAS patterns in the presence of mild-to-moderate coronary artery stenosis is limited. This study aimed to evaluate the incidence and patterns of CAS in patients with insignificant coronary artery stenosis using intracoronary acetylcholine (ACH) provocation test.
View Article and Find Full Text PDFUtility of Cardiac Computed Tomography in the Diagnosis of Apical Hypertrophic Cardiomyopathy: A Case Series.
Heart Views
October 2024
Department of Radiodiagnosis, Postgraduate Institute of Medical Education and Research, Chandigarh, India.
Article Synopsis
- Hypertrophic cardiomyopathy (HCM) is a heart condition where the heart muscle thickens, with apical HCM being a rarer form that affects specific regions of the heart.
- Traditional echocardiography can make diagnosing apical HCM difficult, leading to cases where patients are referred for CT coronary angiography.
- The study presents four cases where CT scans not only ruled out coronary artery disease but also helped confirm apical HCM through detailed evaluation of the CT data.
PHACTR1 and APOC1 genetic variants are associated with multi-vessel coronary artery disease.
Lipids Health Dis
October 2024
Department of Public Health and Epidemiology, Khalifa University of Science and Technology, Abu Dhabi, United Arab Emirates.
Article Synopsis
- Severe coronary artery disease (CAD) is linked to serious complications like heart attacks and angina, and this study focuses on understanding the genetic factors associated with severe and multi-vessel CAD.
- Researchers analyzed data from 1,900 patients with severe CAD and 1,056 healthy controls, identifying 14 genetic variants related to severe disease and finding specific associations based on age.
- Notably, the PHACTR1 gene variant increases the risk for younger individuals, while another variant in APOC1/APOE decreases risk for older adults, highlighting how genetic differences can influence CAD severity and potentially inform personalized treatment strategies.
Association of Angina, Myocardial Infarction and Atrial Fibrillation-A Bidirectional Mendelian Randomization Study.
Br J Hosp Med (Lond)
September 2024
Department of Cardiology, The Affiliated Hospital of Guizhou Medical University, Guiyang, Guizhou, China.
Article Synopsis
- - The study investigates the causal relationship between coronary heart disease (CHD), coronary atherosclerosis, angina, myocardial infarction (MI), and atrial fibrillation (AF) using a method called two-sample Mendelian randomization.
- - Mendelian randomization relies on genetic variations to assess causality, based on three key assumptions: the genetic variant relates to the exposure, is free of confounding factors, and only influences the outcome through that exposure.
- - Findings suggest a genetic link where CAAs, angina, and MI increase the risk of AF, and AF may also heighten the risk of CAAs, highlighting the importance of regular monitoring and appropriate treatment for patients with CHD and AF. *
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