Hemi-infarction of the medulla causes the clinical constellation of symptoms and signs of both the lateral and medial medullary syndromes and nearly always results from occlusion of an intracranial vertebral artery. In the case reported here, with a clinical diagnosis of hemimedullary syndrome, the expected infarction was imaged by magnetic resonance. A review of the literature confirms that the hemimedullary syndrome, in which both medial and lateral syndromes occur simultaneously, is extremely rare, since it yielded only two previous cases with adequate anatomical confirmation.
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http://dx.doi.org/10.1007/BF00314596 | DOI Listing |
Neurology
August 2023
(Abu Dhabi, United Arab Emirates).
Neurology
August 2023
(Dijon, France).
Neurology
March 2023
From the Departments of Neurology (P.K., A.J.), Neurology and Neurointerventional Surgery (S.I.H., S.J.), Neurological Institute, and Department of Neuroradiology (Z.S.), Imaging Sciences and Interventional Radiology Institute, Cleveland Clinic Abu Dhabi, UAE.
Int J Stroke
January 2015
Department of Neurology, Eulji General Hospital, Eulji University, Seoul, Korea.
Neurol Sci
April 2014
Department of Neurology, Guangdong 999 Brain Hospital, Shatai Road, 578, Guangzhou, 510510, China.
Here, we present a rare case of a lateral medullary infarction with ipsilateral hemiparesis, lemniscal sensation loss and hypoglossal nerve palsy. In this case, we proved Opalski's hypothesis by diffusion tensor tractography that ipsilateral hemiparesis in a medullary infarction is due to the involvement of the decussated corticospinal tract. We found that the clinical triad of ipsilateral hemiparesis, lemniscal sensation loss and hypoglossal nerve palsy, which had been regarded as a variant of medial medullary syndrome, turned out to be caused by lateral lower medullary infarction.
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