[Management of femoral neck fracture post total knee arthroplasty].

Zhonghua Yi Xue Za Zhi

Department of Orthopedics, the People's Hospital of Tianjin, Tianjin 300121, China.

Published: April 2011

Objective: To investigate the risk factors, management and prognosis of femoral neck fracture post total knee arthroplasty (TKA).

Methods: From January 2003 to August 2009, 45 cases of femoral neck fracture post TKA were treated. There were 8 males and 37 females with an age range of 56 - 81 years old. Among them, there were ipsilateral (n = 36) and contralateral (n = 9) femoral neck fractures. Due to the fracture site and type, the treatment plans were formulated. And a follow-up was conducted to record the Harris hip score and complications.

Results: The incidence of femoral neck fracture following TKA and osteoporosis had a linear correlation. And the fracture rate increased with the aggravation of osteoporosis. The stability of ipsilateral fracture group was lower than those of contralateral fracture and no fracture groups. Except for 4 mortality cases, 41 patients were followed up for 2 to 7 years with an average period of 3.6 years. There were 12 patients in the cannulated screw fixation group. The outcomes were fracture healing (n = 5) and femoral head necrosis (n = 7). Twenty-five cases underwent femoral head replacement. And 3 died and second fracture occurred in 3 cases. Eight cases underwent total hip arthroplasty. And 1 died and there was 1 case of second fracture. The Harris scores of the cannulated screw fixation group was significantly lower than the hip replacement groups (q test, P < 0.05).

Complications: After internal fixation, 7 cases suffered femoral head necrosis and underwent femoral head replacement. And secondary fractures after hip replacement occurred in 4 cases and they were treated by plate and cable or LISS (less invasive stabilization system) system.

Conclusion: Femoral neck fracture following TKA is usually caused by a low-energy injury. Osteoporosis is one of its high-risk factors. Knee instability is associated with ipsilateral femoral neck fracture. Clinically it can be treated by internal fixation or hip arthroplasty. And internal fixation of femoral neck fracture frequently induces femoral head necrosis. And femoral head replacement is applicable.

Download full-text PDF

Source

Publication Analysis

Top Keywords

femoral neck
32
neck fracture
28
femoral head
24
fracture
15
femoral
13
fracture post
12
head necrosis
12
head replacement
12
internal fixation
12
neck
8

Similar Publications

Background: Total hip arthroplasty is the preferred treatment for advanced hip osteoarthritis, yet complications like hip dislocation (0.2 %-10 %) persist due to factors such as implant design, positioning, surgical technique, and patient-specific conditions. Impingement between prosthetic components or the acetabulum and proximal femur is a primary cause of instability.

View Article and Find Full Text PDF

Background: Venous thromboembolism (VTE) is a common complication after hip arthroplasty. Here, we investigated the clinical efficacy and safety of prophylactic aspirin vs. conventional therapy in hip arthroplasty for femoral neck fracture.

View Article and Find Full Text PDF

Background: Injectable depot medroxyprogesterone acetate (DMPA) is the most common contraceptive choice among young women in Uganda, where HIV burden is high and HIV pre-exposure prophylaxis (PrEP) may be offered. For young women who choose to use both agents concurrently, it is unknown whether they will experience declines in BMD beyond those elicited by either product singly.

Methods: From 2018-2022, we conducted a 2-year prospective study with women ages 16-25 years in Kampala, Uganda desiring pregnancy and HIV prevention.

View Article and Find Full Text PDF

Incident Vertebral Fractures During Romosozumab Treatment in a Patient With a Pathogenic Variant.

JCEM Case Rep

January 2025

Department of Internal Medicine, Erasmus Medical Center, University Medical Center, 3015 CE, Rotterdam, the Netherlands.

A defect in the canonical Wnt-β-catenin pathway may lead to reduced bone strength and increased fracture risk. Sclerostin is a key inhibitor of this pathway by binding to low-density lipoprotein (LDL) receptor-related protein , thereby reducing bone formation. The effectiveness of romosozumab, a human monoclonal antibody that binds sclerostin and prevents this inhibitory effect, has been questioned in patients with inactivating genetic variants in or .

View Article and Find Full Text PDF

Want AI Summaries of new PubMed Abstracts delivered to your In-box?

Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!