Severity: Warning
Message: file_get_contents(https://...@pubfacts.com&api_key=b8daa3ad693db53b1410957c26c9a51b4908&a=1): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests
Filename: helpers/my_audit_helper.php
Line Number: 176
Backtrace:
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 176
Function: file_get_contents
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 250
Function: simplexml_load_file_from_url
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 3122
Function: getPubMedXML
File: /var/www/html/application/controllers/Detail.php
Line: 575
Function: pubMedSearch_Global
File: /var/www/html/application/controllers/Detail.php
Line: 489
Function: pubMedGetRelatedKeyword
File: /var/www/html/index.php
Line: 316
Function: require_once
The acetylcholinesterase activity in selected tissues of prawn, Metapenaeus monoceros showed a significant inhibition during commercial (CgC) and technical (TgC) grade carbaryl exposure. The rate of inhibition of AChE in nervous and non-nervous tissues is more under CgC over TgC exposure, suggests the involvement of emulsifier system for easy penetration of CgC molecule, which is absent in TgC. A progressive recovery of AChE activity from TgC and CgC induced inhibition was in the tissues within 10 days after the transfer of prawns to toxicant free water i.e., recovery or reclamation period. From the study, it has been observed that the inhibition of AChE activity is still persists evenafter 10 days of reclamation period, gives the idea of carbaryl accumulation at the cellular level or the reclamation period is not sufficient for the restoration of normalcy of AChE activity.
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