A glioblastoma multiforma developed 30 years after a penetrating craniocerebral injury in the left parietal region caused by fragments of an artillery projectile. The 3 cm large bone defect was located directly above the tumour. There were close scarry connections between dura, brain and tumour. Partial removal could not prevent the lethal exitus. Causal connection with the accident is assumed to exist.
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MMW Fortschr Med
September 2004
Klinik und Poliklinik für Psychiatrie und Psychotherapie, Universitätsklinikum Münster.
Cranial injuries appear as neurological symptoms such as unconsciousness and focal neurological deficits. Mild cranio-cerebral traumas, that is, brain injuries without a serious neurotraumatologic symptom complex occuroften in practice. Nevertheless, even slight injuries, above all in the frontal cortical brain areas, can be accompanied by characteristic psycho-organic symptoms ("mild cognitive impairment" in the form of a "pseudoneurasthenic syndrome").
View Article and Find Full Text PDFZh Nevropatol Psikhiatr Im S S Korsakova
November 1990
Integrative activity of the brain (psycho-vegeto-somatic+ regulation of functions) was studied in 50 persons with sequelae of grave craniocerebral injury (GCCI). In the acute period of CCI, 19 patients were diagnosed to have brain contusion of severe degree and 31 brain compression. CCI standing had been 1.
View Article and Find Full Text PDFA glioblastoma multiforma developed 30 years after a penetrating craniocerebral injury in the left parietal region caused by fragments of an artillery projectile. The 3 cm large bone defect was located directly above the tumour. There were close scarry connections between dura, brain and tumour.
View Article and Find Full Text PDFA dynamic study of the arteriovenous oxygen and lactic acid content difference, as well as a study of the CSF content of the lactic acid in patients with a severe craniocerebral trauma permitted to evaluate the severity of the pathological process and the results of its treatment. A growing production of lactic acid by the brain and its progressive accumulation in the CSF to a high level as 45-50 mg%, and a sharp dissociation between the increasing lactate level in the CSF and its growing concentration in the venous blood flowing from the brain is a prognostic sign of an unfavourable course and outcome of the disease.
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