Recent research has shown that suppressing food related thoughts can cause a subsequent increase in consumption relative to groups not suppressing, or thinking about food. The present study examined whether the effects of thought suppression on subsequent eating behaviour would interact with participants restrained eating status. One hundred and sixteen female participants were split into three groups. One third suppressed thoughts of chocolate, one third thought about chocolate and the final third thought about anything they wished. Following this, participants took part in a task where they rated two brands of chocolate on several taste characteristics. Participants were unaware that the dependent variable was the amount of chocolate consumed and not taste preference. Participants also completed measures of dietary restraint, craving, guilt and thought suppression. Results indicated that restrained eaters in the suppression condition consumed significantly more chocolate than restrained eaters in the expression or control condition. Participants low on restraint ate statistically equivalent amounts in all three groups. In addition, participants reporting frequent use of thought suppression (assessed by the White Bear Suppression Inventory) reported greater chocolate cravings.
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http://dx.doi.org/10.1016/j.appet.2010.02.001 | DOI Listing |
Int J Mol Sci
December 2024
Centre for Heart Research, The Westmead Institute for Medical Research, The University of Sydney, Westmead, NSW 2145, Australia.
Programmed cell death, especially programmed necrosis such as necroptosis, ferroptosis, and pyroptosis, has attracted significant attention recently. Traditionally, necrosis was thought to occur accidentally without signaling pathways, but recent discoveries have revealed that molecular pathways regulate certain forms of necrosis, similar to apoptosis. Accumulating evidence indicates that programmed necrosis is involved in the development of various diseases, including myocardial ischemia-reperfusion injury (MIRI).
View Article and Find Full Text PDFInt J Environ Res Public Health
November 2024
School of Nursing, Vanderbilt University, Nashville, TN 62374, USA.
HIV care engagement and antiretroviral therapy (ART) adherence interventions aimed at decreasing viral suppression disparities for women living with HIV (WLWH) in the Southern United States (i.e., the South) are few and seldom consider diverse social locations.
View Article and Find Full Text PDFAm J Hum Genet
January 2025
Department of Medicine, Indiana University School of Medicine, Indianapolis, IN, USA.
Each human genome has approximately 5 million DNA variants. Even for complete loss-of-function variants causing inherited, monogenic diseases, current understanding based on gene-specific molecular function does not adequately predict variability observed between people with identical mutations or fluctuating disease trajectories. We present a parallel paradigm for loss-of-function variants based on broader consequences to the cell when aberrant polypeptide chains of amino acids are translated from mutant RNA to generate mutated proteins.
View Article and Find Full Text PDFPsychophysiology
January 2025
Fudan Institute on Ageing, Fudan University, Shanghai, China.
It is common to make risky decisions under time pressure. However, there are ongoing debates regarding the interpretation of the intrinsic mechanisms through which time pressure influences risky decision-making. The current study, combining a sequential risk-taking task, behavioral modeling, and time-resolved multivariate pattern analysis on electroencephalography signals, explored the intrinsic mechanisms underlying the influence of time pressure on risky decision-making.
View Article and Find Full Text PDFProc Natl Acad Sci U S A
January 2025
Department of Psychology, University of York, York YO10 5DD, United Kingdom.
Sleep disturbances are associated with intrusive memories, but the neurocognitive mechanisms underpinning this relationship are poorly understood. Here, we show that sleep deprivation disrupts prefrontal inhibition of memory retrieval, and that the overnight restoration of this inhibitory mechanism is associated with time spent in rapid eye movement (REM) sleep. The functional impairments arising from sleep deprivation are linked to a behavioral deficit in the ability to downregulate unwanted memories, and coincide with a deterioration of deliberate patterns of self-generated thought.
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