Download full-text PDF |
Source |
|---|
Publication Analysis
Top Keywords
Similar Publications
Intramyocellular lipid use is altered with exercise in males with childhood-onset obesity despite no differences in substrate oxidation.
Int J Obes (Lond)
January 2025
Department of Health, Kinesiology, and Applied Physiology, Concordia University, Montreal, QC, Canada.
Objective: Compared to adulthood-onset obesity (AO), those with childhood-onset obesity (CO) are at greater risk of metabolic disease. However, the differences between these two obesity phenotypes are not clear. The aim of this study is to investigate how the age of obesity onset (CO vs.
View Article and Find Full Text PDFObjective: This study aimed to compare physiological changes when wearing a N95 and surgical mask at rest.
Methods: 42 subjects aged 18-55 (19 males; age 22.98 ± 3.
The Epigenetic Machinery and Energy Expenditure: A Network to Be Revealed.
Genes (Basel)
January 2025
Fondazione IRCSS Ca' Granda Ospedale Maggiore Policlinico, 20122 Milano, Italy.
Mendelian disorders of the epigenetic machinery (MDEMs) include a large number of conditions caused by defective activity of a member of the epigenetic machinery. MDEMs are characterized by multiple congenital abnormalities, intellectual disability and abnormal growth. that can be variably up- or down-regulated.
View Article and Find Full Text PDFQuantitative interpretation and modelling of continuous nonprotein respiratory quotients.
Am J Physiol Endocrinol Metab
January 2025
Human and Animal Physiology, Wageningen University, Wageningen, The Netherlands.
The Respiratory Exchange Ratio (RER), which is the ratio of total carbon dioxide produced over total oxygen consumed, serves as a qualitative measure to determine the substrate usage of a particular organism on the whole-body level. Quantification of RER by its direct conversion into %Glucose (%G) and %Lipid oxidation (%L) at a given timepoint can be done by utilizing nonprotein respiratory quotient tables. These tables, however, are limited to specific increments, and intermediate RER values are not covered by these tables.
View Article and Find Full Text PDFEndothelial autophagy-related gene 7 contributes to high fat diet-induced obesity.
Mol Metab
January 2025
Department of Medicine, Division of Endocrinology, Diabetes, and Metabolism, University of Alabama at Birmingham, Birmingham, AL 35294, USA; UAB Comprehensive Diabetes Center, USA. Electronic address:
Objective: Obesity-associated metabolic dysfunction is a major public health concern worldwide. Endothelial dysfunction is a hallmark of metabolic dysfunction, and endothelial cells affect metabolic functions. Because autophagy-related gene 7 (ATG7) is involved in various cellular physiology, we investigated the roles of endothelial cell-ATG7 (EC-ATG7) on high-fat diet-induced obesity and its related metabolic dysfunction.
View Article and Find Full Text PDFWant AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!