Pigment retinal dystrophy induced by the administration of monoiodacetic acid was characterized by a unilateral inhibition of parameters of all components of not only electroretinography, but also evoked potentials of the visual cortex and superior colliculus: higher thresholds of their formation relatively to the controls, and displacement of saturation areas towards low intensities. Correlations of retinogeniculocortical and retinocolliculocortical pathways of visal information transmission were ascertained to be an important factor in the mechanism of visual analyzer functioning in certain abnormalities.
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