Severity: Warning
Message: file_get_contents(https://...@gmail.com&api_key=61f08fa0b96a73de8c900d749fcb997acc09&a=1): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests
Filename: helpers/my_audit_helper.php
Line Number: 176
Backtrace:
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 176
Function: file_get_contents
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 250
Function: simplexml_load_file_from_url
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 1034
Function: getPubMedXML
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 3152
Function: GetPubMedArticleOutput_2016
File: /var/www/html/application/controllers/Detail.php
Line: 575
Function: pubMedSearch_Global
File: /var/www/html/application/controllers/Detail.php
Line: 489
Function: pubMedGetRelatedKeyword
File: /var/www/html/index.php
Line: 316
Function: require_once
The placentas taken from 83 pregnant women with gestation of 37-40 weeks were morphologically and morphometrically studied. Group 1 comprised 23 pregnant women with pulmonary tuberculosis. Group 2 included 40 pregnant women whose pregnancy was complicated by gestosis. Group 3 (control) consisted of 20 healthy pregnant women. Group 1 was found to have chronic placental insufficiency caused by abnormal placental villous immaturity the basis of which was impaired processes of villous differentiation and terminal villous angiogenesis. In most cases, chronic placental insufficiency was compensatory in all the groups under study; but there were differences in compensatory mechanisms. The leading mechanism was cellular in Groups 1 and 2, the mixed (vascular and cellular) types of compensation was predominant in Group 3 (control).
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