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http://dx.doi.org/10.1001/jama.1991.03470200046030DOI Listing

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Background: HIV-associated tuberculosis is a common coinfection in Sub-Saharan Africa, which causes high morbidity and mortality. A sub-set of HIV-associated tuberculosis patients require prolonged hospital admission, during which antiretroviral therapy initiation may be required. The aim of this study was to document the causes of clinical deterioration of hospitalised patients with HIV-associated tuberculosis starting antiretroviral therapy in order to inform healthcare practice in low- to middle-income countries.

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Article Synopsis
  • There is a connection between HIV infection and a higher risk of venous thromboembolic disorders.
  • Some HIV patients experience a hypercoagulable state due to various abnormalities.
  • A specific case is noted where an HIV patient had venous thromboembolism despite normal CD4 levels, with decreased protein C and S and elevated serum homocysteine levels.
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Prevention and treatment of venous thromboembolism are gaining attention in the lay and medical communities because of an increase in frequency, cost, and risk factors. Evidence shows that patients with human immunodeficiency virus (HIV) have multiple risk factors and a 2-10-fold increased risk for venous thromboembolism compared with the general population. A higher rate of venous thromboembolism also occurs in patients with HIV who are younger than 50 years (3.

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