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| http://dx.doi.org/10.1016/s0828-282x(09)70079-0 | DOI Listing |
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Brugada Syndrome and Kawasaki Disease in a Two-Year-Old: A Case of Double Jeopardy.
Cureus
December 2024
Pediatric Cardiology, Children's Hospital at Montefiore, Bronx, USA.
Brugada syndrome (BrS) is a genetic channelopathy that may predispose to ventricular arrhythmia. It is inherited as an autosomal dominant pattern with incomplete penetrance. Fever can unmask Brugada syndrome in children who have a genetic predisposition.
View Article and Find Full Text PDFA Tale of Two Conditions: Pediatric Brugada Syndrome Unveiled-Navigating the Challenges of Coexisting Arrhythmia and Fever-Induced ECG Pattern.
Ann Noninvasive Electrocardiol
January 2025
Department of Paediatrics and Adolescent Medicine, The University of Hong Kong, Pok Fu Lam, Hong Kong.
Background: Brugada syndrome (BrS) is an inherited channelopathy characterized by right precordial ST-segment elevation. This study investigates the clinical and genetic characteristics of children with BrS in Hong Kong.
Methods: A retrospective review was conducted at the only tertiary pediatric cardiology center in Hong Kong from 2002 to 2022, including all pediatric BrS patients under 18 years old.
Impact of hypokalemia on Brugada syndrome: case report unveiling mechanisms beyond QT interval prolongation.
Egypt Heart J
October 2024
Department of Cardiology and Vascular Medicine, Faculty Medicine, Gorontalo State University,, Jalan Jend, Sudirman No.6, Dulalowo Kecamatan Kota Tengah Kota, Gorontalo, 96128, Indonesia.
Article Synopsis
- Brugada syndrome (BrS) increases the risk of sudden death due to dangerous heart rhythms and is characterized by specific ECG changes, notably ST-segment elevation in right precordial leads.
- A case involving a 51-year-old man with low potassium levels (hypokalemia) revealed that while treatment normalized potassium, the Brugada pattern on ECG remained unchanged, and his corrected QT interval was shorter during hypokalemia.
- This case suggests that hypokalemia can reveal the Brugada type-1 pattern without changing it, indicating that further research is needed to explore how hypokalemia could influence ventricular arrhythmias beyond just QT interval changes.
Ajmaline infusion for unmasking short-coupled Purkinje ectopy: Need for speed?
Heart Rhythm
August 2024
Department of Cardiology, UZ Leuven, Leuven, Belgium; Department of Cardiovascular Sciences, UZ Leuven, Leuven, Belgium. Electronic address:
Diagnosis of Brugada Syndrome With a Sodium-Channel-Blocker Test: Who Should Be Tested? Who Should Not?
Circulation
August 2024
Amsterdam University Medical Center, The Netherlands (A.S.A., A.A.W.).
Intravenous infusion of sodium-channel blockers (SCB) with either ajmaline, flecainide, procainamide, or pilsicainide to unmask the ECG of Brugada syndrome is the drug challenge most commonly used for diagnostic purposes when investigating cases possibly related to inherited arrhythmia syndromes. For a patient undergoing an SCB challenge, the impact of a positive result goes well beyond its diagnostic implications. It is, therefore, appropriate to question who should undergo a SCB test to diagnose or exclude Brugada syndrome and, perhaps more importantly, who should not.
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