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Correction: Montalesi et al. Divergent Effects of Daidzein and Its Metabolites on Estrogen-Induced Survival of Breast Cancer Cells. 2020, , 167.
Cancers (Basel)
November 2024
Department of Science, University Roma Tre, Viale Guglielmo Marconi 446, I-00146 Roma, Italy.
In the original publication [...
View Article and Find Full Text PDFSimulated Galactic Cosmic Radiation Exposure-Induced Mammary Tumorigenesis in Mice Coincides with Activation of ERα-ERRα-SPP1 Signaling Axis.
Cancers (Basel)
November 2024
Department of Oncology, Lombardi Comprehensive Cancer Center, Georgetown University Medical Center, Washington, DC 20057, USA.
Background: Exposure to galactic cosmic radiation (GCR) is a breast cancer risk factor for female astronauts on deep-space missions. However, the specific signaling mechanisms driving GCR-induced breast cancer have not yet been determined.
Methods: This study aimed to investigate the role of the estrogen-induced ERα-ERRα-SPP1 signaling axis in relation to mammary tumorigenesis in female mice exposed to simulated GCR (GCRsim) at 100-110 days post-exposure.
Gut microbiota and breast cancer: systematic review and meta-analysis.
Breast Cancer
December 2024
Laboratory of Translational Biomedicine, Universidade do Extremo Sul Catarinense, Criciuma, SC, 88806-000, Brazil.
Background: The gastrointestinal microbiota can modulate systemic estrogens, potentially influencing estrogen-induced breast neoplasia development. This study aimed to assess alterations in the gut microbiota in breast cancer patients.
Methods: A search strategy was developed using the terms: "Microbiota," "Gastrointestinal Microbiome," "Breast Cancer," and synonyms.
Analysis of the Abasic Sites in Breast Cancer Patients With 5 Year Postoperative Treatment Without Recurrence in Taiwan.
Cancer Control
November 2024
Department of Environmental Engineering, National Chung Hsing University, Taichung, Taiwan.
Purpose: This prospective study aimed to investigate estrogen-induced carcinogenesis by assessing the background levels of abasic sites (apurinic/apyrimidinic sites, AP sites) in Taiwanese breast cancer patients following 5 years of postoperative treatment without recurrence (5-year survivors) (n = 70). The study also sought to compare the extent of these DNA lesions with those found in healthy controls and in breast cancer patients prior to treatment.
Methods: Abasic sites were measured using an aldehyde reactive probe and quantified as the total number of abasic sites per total nucleotides.
SHP2-Triggered Endothelial Cell Activation Fuels Estradiol-Independent Endometrial Sterile Inflammation.
Adv Sci (Weinh)
November 2024
State Key Laboratory of Pharmaceutical Biotechnology and Nanjing Drum Tower Hospital the Affiliated Hospital of Nanjing University Medical School, School of Life Sciences, Nanjing University, 163 Xianlin Avenue, Nanjing, 210023, China.
Sterile inflammation occurs in various chronic diseases due to many nonmicrobe factors. Examples include endometrial hyperplasia (EH), endometriosis, endometrial cancer, and breast cancer, which are all sterile inflammation diseases induced by estrogen imbalances. However, how estrogen-induced sterile inflammation regulates EH remains unclear.
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