Download full-text PDF |
Source |
|---|---|
| http://dx.doi.org/10.1002/dmr.5610070206 | DOI Listing |
Publication Analysis
Top Keywords
Similar Publications
Integrating New Approach Methodologies to Address Environmental Pancreatic Toxicity and Metabolic Disorders.
Biology (Basel)
January 2025
Center for Computational Toxicology and Exposure, US Environmental Protection Agency, Research Triangle Park, NC 27711, USA.
Advancing our understanding of pancreatic toxicity and metabolic disorders caused by environmental exposures requires innovative approaches. The pancreas, a vital organ for glucose regulation, is increasingly recognized as a target of harm from environmental chemicals and dietary factors. Traditional toxicological methods, while foundational, often fail to address the mechanistic complexities of pancreatic dysfunction, particularly under real-world conditions involving multiple exposures.
View Article and Find Full Text PDFPancreaticoduodenectomy in trauma patients.
Trauma Surg Acute Care Open
January 2025
Surgery, University of Maryland Baltimore, Baltimore, Maryland, USA.
A rare cause of recurrent pancreatitis: intraductal papillary mucinous neoplasm in the duct of Santorini (with video).
Gastrointest Endosc
October 2024
Department of Gastroenterology, Qilu Hospital of Shandong University, Jinan, Shandong, China. Electronic address:
Autophagy and its role in gastrointestinal diseases.
World J Gastroenterol
September 2024
Department of Gastroenterology, First Medical Center of Chinese People's Liberation Army General Hospital, Beijing 100853, China.
Article Synopsis
- Gastrointestinal disorders include various conditions affecting the digestive organs, such as the esophagus, stomach, and intestines, with a particular focus on the role of autophagy in these diseases.
- The paper examines how autophagy influences inflammation-related disorders like ulcerative colitis and Crohn's disease, as well as gastrointestinal cancers like esophageal and colorectal cancer.
- The review emphasizes the complexity of autophagy in gastrointestinal diseases and stresses the need for further research to fully realize its therapeutic potential.
Commentary on "Intra-islet α-cell Gs signaling promotes glucagon release".
J Diabetes Investig
December 2024
Metabolic Signal Research Center, Institute for Molecular and Cellular Regulation, Maebashi, Japan.
The Gs receptor in pancreatic α cells promotes glucagon transcription and secretion via increasing cAMP. The Gi receptor in pancreatic α cells suppresses glucagon secretion via decreasing cAMP. The Gq receptor in pancreatic α cells promotes glucagon secretion via releasing Ca from ER to cytoplasm.
View Article and Find Full Text PDFWant AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!